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Titlebook: HDL Deficiency and Atherosclerosis; G. Assmann Book 1995 Kluwer Academic Publishers 1995 Lipoprotein.atherosclerosis.cardiovascular.corona

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31#
發(fā)表于 2025-3-26 21:28:14 | 只看該作者
32#
發(fā)表于 2025-3-27 03:31:08 | 只看該作者
33#
發(fā)表于 2025-3-27 09:05:12 | 只看該作者
Gene therapy for the genetic dyslipoproteinaemias,idated. This new information provides a conceptual framework for understanding lipoprotein metabolism in normal subjects and patients with the genetic dyslipoproteinaemias. Schematic overviews of our current concepts of lipoprotein metabolism are illustrated in Figs. 1 and 2.
34#
發(fā)表于 2025-3-27 10:41:24 | 只看該作者
https://doi.org/10.1007/978-94-011-6585-3Lipoprotein; atherosclerosis; cardiovascular; coronary heart disease; gene therapy; genetics; heart; heart
35#
發(fā)表于 2025-3-27 16:15:40 | 只看該作者
36#
發(fā)表于 2025-3-27 18:58:04 | 只看該作者
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發(fā)表于 2025-3-28 03:01:33 | 只看該作者
39#
發(fā)表于 2025-3-28 07:35:04 | 只看該作者
The Future of Predictive Safety EvaluationapoA-I deficiency, LCAT deficiency, and fish-eye disease, but also patients with Tangier disease or unclassified forms of HDL deficiency did not present with premature atherosclerosis.. Family histories of these patients did not indicate any increased prevalence of coronary heart disease (CHD) event
40#
發(fā)表于 2025-3-28 10:52:07 | 只看該作者
https://doi.org/10.1057/9780230606906 a target-specific flux of cholesterol from intracellular pools to the plasma membrane are only poorly understood at present. It is generally accepted that intracellular trafficking of cholesterol and other sterols is not mediated by non-specific diffusion but requires target-specific transport mech
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