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Titlebook: Virus Variability, Epidemiology and Control; Edouard Kurstak,R. G. Marusyk,M. H. V. Regenmortel Book 1990 Springer Science+Business Media

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書目名稱Virus Variability, Epidemiology and Control
編輯Edouard Kurstak,R. G. Marusyk,M. H. V. Regenmortel
視頻videohttp://file.papertrans.cn/984/983518/983518.mp4
叢書名稱Applied Virology Research
圖書封面Titlebook: Virus Variability, Epidemiology and Control;  Edouard Kurstak,R. G. Marusyk,M. H. V. Regenmortel Book 1990 Springer Science+Business Media
描述Virus Variability and Impact on Epidemiology and Control of Diseases E. Kurstak and A. Hossain I. INTRODUCTION An important number of virus infections and their epidemic developments demonstrate that ineffec- tiveness of prevention measures is often due to the mutation rate and variability of viruses (Kurstak et al., 1984, 1987). The new human immunodeficiency retroviruses and old influenza viruses are only one among several examples of virus variation that prevent, or make very difficult. the production of reliable vaccines. It could be stated that the most important factor limiting the effectiveness of vaccines against virus infections is apparently virus variation. Not much is, how- ever, known about the factors influencing and responsible for the dramatically diverse patterns of virus variability. II. MUTATION RATE AND VARIABILITY OF HUMAN AND ANIMAL VIRUSES Mutation is undoubtedly the primary source of variation, and several reports in the literature suggest that extreme variability of some viruses may be a consequence of an unusually high mutation rate (Holland et al., 1982; Domingo et al., 1985; Smith and Inglis, 1987). The mutation rate of a virus is defined as the probabil
出版日期Book 1990
關(guān)鍵詞Antigen; avian influenza; development; epidemiology; evolution; immunodeficiency; infection; infections; mol
版次1
doihttps://doi.org/10.1007/978-1-4757-9271-3
isbn_softcover978-1-4757-9273-7
isbn_ebook978-1-4757-9271-3
copyrightSpringer Science+Business Media New York 1990
The information of publication is updating

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Evolutionary Lineages and Molecular Epidemiology of Influenza A, B, and C Virusescells, an additional protein (NB) coded for by the neuraminidase gene is found. In contrast to influenza A and B viruses, influenza C viruses contain only seven RNA segments encoding eight known proteins, and the C viruses lack the gene coding for the neuraminidase.
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Analysis of Rotavirus Proteins by Gene Cloning, Mutagenesis, and Expressionthat a better understanding of the molecular features of these proteins might lead to the development of second-generation recombinant vaccines. Although the general feasibility of developing recombinant rotavirus vaccines has yet to be established, this might occur through the development of engine
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Book 1990he primary source of variation, and several reports in the literature suggest that extreme variability of some viruses may be a consequence of an unusually high mutation rate (Holland et al., 1982; Domingo et al., 1985; Smith and Inglis, 1987). The mutation rate of a virus is defined as the probabil
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morphic to the group .[.] of automorphisms . of the polynomial ring ?[.] by means of the correspondence .(.) = . where .(..) = ..(.). Polynomial maps .(.) satisfying det .(.) = . ≠ 0 are called .. We can and do assume that .(0) = 0 and .(0) = .. Five main problems arise:
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John M. Coffin presence of each other and with the external environment. This included the study of electromagnetic compatibility, as systems and components started to have influence upon each other just due to their operation. EMC developed over the years, and has become a specialized area of engineering applica
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