Titlebook: Uremic Toxins; Severin Ringoir,Raymond Vanholder,Shaul G. Massry Book 1987 The Editor(s) (if applicable) and The Author(s), under exclusiv

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Middle Molecules and the 7 C Factor,designed to increase or decrease the presumed level of middle molecules (MM) in the body fluids relating these changes to the symptomatology of the patients or to the in vitro toxicity of plasma and dialysate (see references in 1). However, most of these studies were inconclusive and no direct deter

群島

老人病學(xué)

Parallel

Roles of Hippurate and Indoxyl Sulfate in the Impaired Ligand Binding by Azotemic Plasma,er, impaired plasma protein binding of acidic drugs and endogenous metabolites, hoping to discover investigative approaches which might be applicable to study of more complex uremic disorders. Our initial interest in the problem arose from our finding of markedly reduced levels of plasma tryptophan

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狂亂

特征

The Role of Lipids in Progressive Glomerular Disease, acid and mevinolin. Pharmacologically, these two agents have different mechanisms of action. Clofibric acid affects both cholesterol and triglyceride metabolism, while mevinolin inhibits 3-hydroxy-3 methyl- glutaryl coenzyme A reductase, the rate limiting enzyme in cellular cholesterol synthesis. I

Density

Aluminum an Uremic Toxin, aluminum is virtually universal as aluminum constitutes a substantial part of the earth’s crust (8%) and is found in food, medicine and cosmetics. Besides this aluminum has a lot of industrial applications. Until recently, aluminum was generally considered to be a relatively nontoxic metal, which i

instructive

ornithology

Pathogenesis and Consequences of the Alteration of Glucose Metabolism in Renal Insufficiency,est pointed to the abnormal liver glycogen metabolism and its decreased liver concentration (Cohen, 1962; Westervelt and Schreiner, 1962). However, later on the glucagon and epinephrine studies have not been confirmed and normal liver glycogen concentration was found in a group of patients with norm