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Titlebook: Urate Deposition in Man and its Clinical Consequences; Ursula Gresser,Nepomuk Z?llner Conference proceedings 1991 Springer Verlag, Berlin

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書目名稱Urate Deposition in Man and its Clinical Consequences
編輯Ursula Gresser,Nepomuk Z?llner
視頻videohttp://file.papertrans.cn/944/943845/943845.mp4
圖書封面Titlebook: Urate Deposition in Man and its Clinical Consequences;  Ursula Gresser,Nepomuk Z?llner Conference proceedings 1991 Springer Verlag, Berlin
描述One person in four in the industrialized countries suffersfrom hyperuricemia and is therefore at risk of developinggouty arthritis, nephrolithiasis, or any of the otherconsequences of uratedeposition.At present, far too little is known about urate depositionand the mechanisms by which it occurs, as well as aboutitsclinical consequences, which include formation of toph; overthehelix of the ear or in bones close to joints that havenever exhibited an attack, development of bursitis, chronictendovaginitis leading to carpal tunnel syndrome, and goutyparaplegia. Information on these matters isneeded toestimate the risks of hyperuricemia and to determinewhentherapeutic intervention is indicated. The contributions anddiscussions in this book, resulting from an internationalsymposium held in December 1990 in the MedizinischePoliklinik in Munich, provide anup-to-date source ofcurrent knowledge about hyperuricemia in man and itsclinical consequences.
出版日期Conference proceedings 1991
關鍵詞Hyperurik?mie; Nierenstein; development; ear; intervention; joint; kidney; metabolism; renal insufficiency; m
版次1
doihttps://doi.org/10.1007/978-3-642-84491-1
isbn_softcover978-3-642-84493-5
isbn_ebook978-3-642-84491-1
copyrightSpringer Verlag, Berlin Heidelberg 1991
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The Enigma of Urate Deposition, inflammatory cells, with an outer layer of giant cells and epithelial cells. Larger deposits — nodules — are made up of large masses of urate (hydrated monosodium urate) together with acute inflammatory cells and giant cells. Calcium salts are commonly included along with fat, protein and polysacch
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Precipitation and Deposition of Monosodium Urate Monohydrate Crystals in Tissues,stant feature of symptomatic gouty arthritis, whether this is due to inflammation in the acute attack or to destructive arthropathy in tophaceous gout [1]. Hyperuricemia, defined as a serum urate level greater than 7.0 mg/dl, is a very frequent, but not indispensable, requirement for MSU crystal dep
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