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Titlebook: Studies on Hepatic Disorders; Emanuele Albano,Maurizio Parola Book 2015 Springer International Publishing Switzerland 2015 cell signaling.

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樓主: obdurate
21#
發(fā)表于 2025-3-25 05:10:48 | 只看該作者
S. Pelusi,L. Valenti,Silvia Fargionone umbrella at the provincial level. New mechanisms and institutional linkages for generating, evaluating, adapting, and applying location-specific agricultural technologies have been installed. A new paradigm adopting a participatory bottom-up planning approach is being implemented. A number of op
22#
發(fā)表于 2025-3-25 09:20:41 | 只看該作者
23#
發(fā)表于 2025-3-25 13:31:40 | 只看該作者
24#
發(fā)表于 2025-3-25 18:08:26 | 只看該作者
Oxidative Mechanisms in Liver Senescence and Regenerationf oxygen-centered cell metabolism; rather, they are actively generated to help establishing and maintaining the redox balance of the cell..Within this novel conceptual context, this chapter will address the role of ROS signaling in the regulation of the cell cycle and the induction of cell senescenc
25#
發(fā)表于 2025-3-25 22:43:09 | 只看該作者
Oxidative Stress and Liver Inflammationlcoholic liver disease, endotoxin-induced liver disease, viral hepatitis, fibrosis, hepatocellular carcinoma (HCC), toxin-mediated liver injury, and cholestatic disease. Evidence for the role of inflammation and oxidative stress in these conditions and models is presented and discussed, as is the po
26#
發(fā)表于 2025-3-26 02:45:30 | 只看該作者
Oxidative Stress and Liver Ischemia–Reperfusion Injuryration leading to liver injury. The contribution of hepatic lipid composition, particularly lipid composition of mitochondrial membranes determine the increased ROS generation during hepatic I/R due to compromised mitochondrial antioxidant defenses. Accumulating evidence from clinical and experiment
27#
發(fā)表于 2025-3-26 07:35:39 | 只看該作者
28#
發(fā)表于 2025-3-26 12:22:04 | 只看該作者
29#
發(fā)表于 2025-3-26 16:17:47 | 只看該作者
30#
發(fā)表于 2025-3-26 18:36:01 | 只看該作者
Oxidative Stress in Nonalcoholic Fatty Liver Disease in the pathogenesis of NAFLD. Different sources of oxidative stress coexists in hepatocytes especially those derived from mitochondrial, microsomal, peroxisomal, and lysosomal origin, many of them linked to FFA metabolism, as we will discuss in detail.
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