Titlebook: Return of the Crazy Bird; The Sad, Strange Tal Clara Pinto-Correia Book 2003 Springer Science+Business Media New York 2003 Museum.Nation.an

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n preclinical and clinical studies. At present, amantadine, a low-affinity non-competitive NMDA receptor antagonist, represents a recommended add-on agent to decrease the dyskinetic motor complications of the dopaminergic therapy..The chapter will describe advances in basic research, preclinical and

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he major difference that the flux in the pathway producing transmitter glutamate is comparable to the rate of glucose metabolism in brain, whereas that producing transmitter ATP is orders of magnitude smaller than the metabolic turnover between ATP and ADP. Moreover, de novo glutamate production occ

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f these neuropathologies in a beneficial or detrimental way. Nucleotides, e.g. adenosine 5′-triphosphate (ATP), released after acute or chronic neuronal injury, are important mediators of glial activation and astrogliosis..Acute injury may cause significant changes in ATP balance, resulting in (1) a

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administration of glutamate caused loss of neurons in the inner nuclear layer of the retina in both adult and neonatal mice .. Olney extended these findings to other regions of brain, including neurons in the roof of the third ventricle, the hypothalamus, and the dentate gyms .. Changes evolved rap

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administration of glutamate caused loss of neurons in the inner nuclear layer of the retina in both adult and neonatal mice .. Olney extended these findings to other regions of brain, including neurons in the roof of the third ventricle, the hypothalamus, and the dentate gyms .. Changes evolved rap

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