Titlebook: Regulation of Membrane Na+-K+ ATPase; Sajal Chakraborti,Naranjan S Dhalla Book 2016 Springer International Publishing AG, part of Springer

Arthr-

笨重

Mechanics

Na, K-ATPase α4: An Isoform Dedicated to Sperm Functionhis review discusses the particular expression, functional properties, regulation, mechanism of action, and role of Na-K-ATPase α4 in the context of the physiology of the male gamete. The current experimental evidence shows that the appearance of α4 during evolution is not a redundant event but rath

不規(guī)則

惡心

Understanding the Dysfunction of Na+/K+-ATPase in Rapid-Onset Dystonia Parkinsonism and Amyotrophic spinal cord of sporadic and familial amyotrophic lateral sclerosis (ALS) patients. The α2-Na./K.-ATPases are also enriched in astrocytes expressing mutant superoxide dismutase 1 (SOD1), which causes familial ALS. Here we focused to understand the physiological and molecular fundamentals associated

DAUNT

Irrepressible

重畫只能放棄

The Astrocytic Na+, K+-ATPase: Stimulation by Increased Extracellular K+, β-Adrenergic Activation, O and its influence on the enzyme’s kinetic parameters is reviewed together with stimulation of the enzyme by noradrenaline and its functional importance. So are details of Na.,K.-ATPase signaling in response to submicromolar concentrations of ouabain and/or low mM K. concentrations without which the

Magisterial

Regulation of the Cardiac Na+/K+-ATPase by Phospholemmantructures in pre-splicing mRNA. The overall physiological role of cardiac PLM is to protect the heart under conditions of increased heart rate and oxidative stress avoiding calcium overload of the cytoplasm and arrhythmias. The PLM tetramer possibly exists as a storage pool in order for the heart to

insolence

Regulation of Brain Na-K ATPase Activity by Noradrenaline with Particular Reference to Normal and AlMSD-induced elevated NA could modulate Na-K ATPase activity in the brain and thus modulates the neuronal and brain excitability. In this chapter first we discuss the mechanism of increase in NA level in the brain after REMSD. Then we discuss the effect of such elevated NA on neuronal and glial Na-K