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Titlebook: Regulation of Cerebral Blood Flow and Metabolism. Neurosurgical Treatment of Epilepsy. Rehabilitatio; Proceedings of the 3 Rolf Wüllenweber

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31#
發(fā)表于 2025-3-26 23:48:30 | 只看該作者
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發(fā)表于 2025-3-27 04:45:14 | 只看該作者
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發(fā)表于 2025-3-27 09:01:43 | 只看該作者
34#
發(fā)表于 2025-3-27 11:42:03 | 只看該作者
Oxygen Metabolism and Microcirculation of the Brain in Various Conditions: Experimental Investigatiol pressure is considered to provide cerebral autoregu-lation (.), whereas pCO. tension and tissue lactate level are assumed to regulate regional cerebral blood flow (rCBF) according to metabolic requirements (., .). The means by which regulation of local cerebral microcirculation (1CBF) occurs, howe
35#
發(fā)表于 2025-3-27 15:23:58 | 只看該作者
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發(fā)表于 2025-3-27 19:30:31 | 只看該作者
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發(fā)表于 2025-3-28 00:55:47 | 只看該作者
38#
發(fā)表于 2025-3-28 03:19:38 | 只看該作者
A New Experimental Model of Sinus Thrombosis in Ratsy exists as to the appropriate therapy. By contrast with research into arterial cerebrovascular disorders, only a few studies have focused on the venous manifestations (., .). Several experimental approaches (often failures) have been made in larger animals (., .; cf. Table 1), whereas to the best o
39#
發(fā)表于 2025-3-28 08:42:21 | 只看該作者
Pathophysiology of Experimental Brain Infarction. Because of the low energy reserves of the brain, interruption of blood flow rapidly induces metabolic disturbances which may cause considerable functional deficits or even tissue necrosis. In the following, the pathophysiology of these disturbances is described in experimental brain infarcts, prod
40#
發(fā)表于 2025-3-28 10:53:43 | 只看該作者
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