Titlebook: Monetary Integration in Europe; Horst Tomann Book 2007 Horst Tomann 2007 economic policy.fiscal policy.growth.monetary policy.monetary uni

Baleful

書目名稱Monetary Integration in Europe影響因子(影響力)




書目名稱Monetary Integration in Europe影響因子(影響力)學(xué)科排名




書目名稱Monetary Integration in Europe網(wǎng)絡(luò)公開度




書目名稱Monetary Integration in Europe網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Monetary Integration in Europe被引頻次




書目名稱Monetary Integration in Europe被引頻次學(xué)科排名




書目名稱Monetary Integration in Europe年度引用




書目名稱Monetary Integration in Europe年度引用學(xué)科排名




書目名稱Monetary Integration in Europe讀者反饋




書目名稱Monetary Integration in Europe讀者反饋學(xué)科排名

缺陷

珠寶

Studies in Economic Transitionhttp://image.papertrans.cn/m/image/638788.jpg

沙漠

Book 2007Can the European Economic and Monetary Union survive as an institution providing the highest degree of monetary integration? Can it withstand crises in international markets and contribute to the stability of the global financial system? This book addresses these questions, emphasising the need for new forms of economic policy coordination.

HIKE

https://doi.org/10.1057/9780230288621economic policy; fiscal policy; growth; monetary policy; monetary union

PAEAN

膠狀

Research for Control System of Soccer Robot Based on DSP,rocess of the six members constituting the fundamental life system is summarized based on the discussions described in the respective chapters. Successively, two research approaches exploring the origins of six members constituting the fundamental life system or protein, cell structure, metabolism,

obsession

Biochemistry of the Basement Membrane in Diabetes Mellitus,rglycemic animals and humans (Brownlee and Cahill, 1979). Where the effect of treatment has been studied, many, if not all of these abnormalities, are returned towards normal with insulin, and it seems likely that some of these may play a role in the pathogenesis of diabetic renal disease. Others ar

extemporaneous

信條

Urban Services in Developing Countriesspir Med 105:1129–1139, 2011). Mutant forms of AAT play a well-documented role in disease pathogenesis: misfolding of AAT protein, accumulation of misfolded protein polymers, and an associated decrease in secreted, functional monomers are known to cause clinical dysfunction and disease through both