Titlebook: Mechanisms of Viral Pathogenesis; From Gene to Pathoge A. Kohn,P. Fuchs Conference proceedings 1984 Martinus Nijhoff Publishing, Boston 198

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Host Range Transition of Lymphotropic Minute Virus of Mice During Persistent Infection in L-Cells and a subsequent establishment of a short term, 150 day long persistent infection. The virus produced by the carrier culture differed from MVM(i) in its ability to replicate lytically in A9 cells and the parallel loss of infectivity to T-lymphocytes. The newly acquired host range was a stable prope

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Transforming Genes of Retroviruses and Cancer Cellsse viruses have arisen in nature by recombination of replication-competent type C RNA viruses with a limited set of evolutionarily well-conserved cellular genes. When incorporated within the retrovirus genome, such transduced cellular (.) sequences acquire transforming properties. To investigate the

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Pathogenicity of Vesicular Stomatitis Virus is Potentially Mediated by Wild-Type Leader RNA which In-sensitive mutants and defective-interfering viruses indicate that viral transcription is essential for inhibiting cellular nucleic acid synthesis. Inactivation of viral genome functions by UV-irradiation implicated the 47-nucleotide wild-type leader RNA as the potential inhibitor of cellular RNA an

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Structural Feature of Picornavirus RNA Involved in Pathogenesis: A Very High Affinity Binding Site fit binds directly to mRNA. The binding of eIF-2 to mRNA is highly specific and occurs in Mengovirus RNA and satellite tobacco necrosis virus RNA at the nucleotide sequences that constitute the ribosome binding sites. A molecule of Mengovirus RNA competes in cell-free translation 35-fold more strongl

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