Titlebook: Leukocyte Integrins in the Immune System and Malignant Disease; Bernhard Holzmann,Hermann Wagner Book 1998 The Editor(s) (if applicable) a

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VLA-4-Mediated Signaling,inked common β-chain, termed β1 (CD29) (. 1992; . 1993). The VLA mainly function as cell surface receptors mediating cell-to-cell and cell-to-extracellular matrix (ECM) adhesive interactions. They constitute a major class of adhesive receptors expressed by T cells. On resting CD4. T cells, the VLA/C

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,The Roles of α4-Integrins in the Development of Insulin-Dependent Diabetes Mellitus, (PP), then to lymph and back to blood (. and . 1964). Smaller numbers of lymphocytes migrate from blood to extranodal tissues such as pancreas and then through lymphatic vessels to LN (. et al. 1990). An important feature of this migration is the ability of lymphocytes to recognize and adhere to th

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,Immunosuppression by Blocking α4-Integrins/VCAM-1 Adhesion,d by T lymphocytes. The initial step consists of T lymphocyte adhesion to graft endothelium, a process that is mediated by cell adhesion molecules. It has been shown that there are several pathways for recognition of alloantigens on the transplanted vascularized organs, and cell adhesion molecules p

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Virus-Induced T Cell Activation and the Inflammatory Response,anisms regulating the generation and function of effector T cells is central to the study of viral pathogenesis. It is characteristic that T cells — as opposed to B cells — are only able to exert their effector function within a very limited distance. Therefore, once effector T cells are generated,

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,α4 Integrins and Tumor Metastasis,invasion of the adjacent tissue (. et al. 1991; . 1982; . et al. 1993). The migration through the interstitial stroma, mainly comprised of extracellular matrix proteins such as vitronectin, collagens or fibronectin, is followed by entry into the vasculature, either by transport through the lymphatic

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