Titlebook: Human Apolipoprotein Mutants; Impact on Atheroscle C. R. Sirtori,A. V. Nichols,G. Franceschini Book 1986 Springer Science+Business Media Ne

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The Limone Sul Garda GeneBarr et al., 1951; Gofman et al., 1966; Miller & Miller, 1975; Gordon et al., 1977). The mechanism by which they confer protection against atherosclerosis is unclear. . experiments involving isolated erythrocytes and cultured cells suggest that HDL may be involved in the delivery of cholesterol from

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Lipid Binding Properties of Human Apo Al Mutantsharacterized (3,4). As these mutants were detected by isofocusing, they all involve one or more mutations at charged residues. These mutations occur at different locations in the apoprotein sequence so that some of them might influence the apoprotein-lipid association. We have studied the lipid bind

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Analysis of Apolipoprotein A-I Synthesized in Vitro from Chick Intestinal mRNAeins (HDL) (1–5). In all species examined so far apo A-I is synthesized in liver and intestine (6–8) as a precursor molecule (pre-proapo A-I) which is characterized by an ami-noacid extension at its NH terminal end (signal peptide). Part of this sequence is cleaved co-translationally to originate th

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Versatile , Expression-Modification Vectors as Tools for Structure-Function Studies of Apolipoprotei (reviewed by Harris, 1983). One such vector, pAS1, contains the strong, regulatable phage lambda promoter, P., two sites for N utilization, (Nut. and Nut.), and the ribosome binding site and initiation codon of the ell gene (Rosenberg et al., 1983). More recently, a derivative of pAS1 was described

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S.-H. Chen,G. Franceschini,C. R. Sirtori,L. Chanilhaftigkeit einer Investition von den den Kalkulationszinsfu? bestimmenden Finanzierungskosten abh?ngt; umgekehrt entscheiden die Investitionsm?glichkeiten über die Vorteilhaftigkeit der Aufnahme von finanziellen Mitteln. Die Berücksichtigung dieser Interdependenzen erfolgt im Kapitalwertmodell rec