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Titlebook: Handbook of Neurochemistry and Molecular Neurobiology; Degenerative Disease Abel Lajtha (Director),Moussa B. H. Youdim (Profes Reference wo

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發(fā)表于 2025-3-21 17:42:42 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Handbook of Neurochemistry and Molecular Neurobiology
副標題Degenerative Disease
編輯Abel Lajtha (Director),Moussa B. H. Youdim (Profes
視頻videohttp://file.papertrans.cn/422/421748/421748.mp4
圖書封面Titlebook: Handbook of Neurochemistry and Molecular Neurobiology; Degenerative Disease Abel Lajtha (Director),Moussa B. H. Youdim (Profes Reference wo
出版日期Reference work 2007Latest edition
關(guān)鍵詞Degenerative; Diseases; Handbook; Lajtha; Molecular; Nervous; Neurobiology; Neurochemistry; System
版次3
doihttps://doi.org/10.1007/978-0-387-30377-2
isbn_ebook978-0-387-30377-2
copyrightSpringer-Verlag US 2007
The information of publication is updating

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,Drug Treatment of Motor Symptoms in Parkinson’s Disease,ostsynaptic dopaminergic uptake sites. These long‐term motor complications are fluctuations of movement and are mostly associated with levodopa owing to the short half‐life of the drug. They considerably burden quality of life in PD patients and their caregivers. These complications follow the so‐ca
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,Huntington’s Disease: Unraveling the Pathophysiological Cascade Behind a Singular Gene Defect,hold, which leads to formation of β‐strands and polyglutamine aggregation. As a histopathological hallmark, intranuclear huntingtin‐positive aggregates have been identified, making HD another neurodegenerative disease, with the presence of insoluble protein aggregates, like Alzheimer‘s or Parkinson‘
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Multiple Sclerosis and Autoimmune Encephalomyelitis,plex and heterogeneous, involving cytotoxic T‐lymphocytes, specific autoantibodies and toxic products of activated macrophages and microglia cells. The mechanisms of tissue injury, involved in the formation of MS lesions are in part reflected in different models of autoimmune encephalomyelitis. Howe
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