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21#
發(fā)表于 2025-3-25 04:15:51 | 只看該作者
Inhibition of arginine synthesis by urea: A mechanism for arginine deficiency in renal failure whicc acid (ASA) and the hydroxyl radical or SIN-1 which generates superoxide and NO simultaneously. However, an excess of NO, which also serves as a scavenger of the hydroxyl radical, inhibited GSA synthesis. We also reported that arginine, citrulline or ammonia plus ornithine, all of which increase ar
22#
發(fā)表于 2025-3-25 10:15:21 | 只看該作者
23#
發(fā)表于 2025-3-25 12:37:20 | 只看該作者
24#
發(fā)表于 2025-3-25 18:46:30 | 只看該作者
25#
發(fā)表于 2025-3-25 20:32:46 | 只看該作者
The clinical syndrome of creatine transporter deficiency,on Magnetic Resonance Spectroscopy (MRS) indicated an absence of creatine and phosphocreatine in the brain of a male patient characterized by developmental delay, mild epilepsy and severe expressive language impairment. Subsequent genetic testing revealed a defect in the X-linked creatine transporte
26#
發(fā)表于 2025-3-26 02:34:25 | 只看該作者
Creatine supplementation in health and disease: What is the evidence for long-term efficacy?, available literature mainly covers the short-term (one to several weeks) effects of creatine supplementation on skeletal muscle function in health and disease, which is of little help to evaluate the long-term (two or more months) potential of creatine as a drug in chronic disorders, such as neurod
27#
發(fā)表于 2025-3-26 04:19:29 | 只看該作者
28#
發(fā)表于 2025-3-26 11:32:59 | 只看該作者
29#
發(fā)表于 2025-3-26 16:11:25 | 只看該作者
Restricted neuronal expression of ubiquitous mitochondrial creatine kinase: Changing patterns in deitochondrial creatine kinase (uMtCK) supports energy metabolism in all neurons. To explore the possibility that uMtCK expression in neurons is modulated by activity and during development, we used immunocytochemistry to detect uMtCK-containing mitochondria. In the adult brain, subsets of neurons inc
30#
發(fā)表于 2025-3-26 16:49:01 | 只看該作者
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