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21#
發(fā)表于 2025-3-25 04:55:49 | 只看該作者
Morphological and Behavioral Effects of Basic Fibroblast Growth Factor and Heparin on Transplanted in vitro as a trophic factor for certain fetal CNS cells and for survival of fetal astrocytes. In vivo bFGF has a preserving effect on damaged adult septal cells. The present experiments were performed to investigate the effect of heparin and the combination of heparin with bFGF on the functional re
22#
發(fā)表于 2025-3-25 08:20:18 | 只看該作者
23#
發(fā)表于 2025-3-25 12:52:12 | 只看該作者
Synthetic Peptide Agonists and Antagonists of Basic Fibroblast Growth Factor: Increasing Cellular Sntial as a therapeutic agent in Alzheimer’s disease or other neurologic diseases. Unlike nerve growth factor (NGF), bFGF is a multifunctional growth factor with additional effects on glial cells, endothelial cells, and fibroblasts. The actions of bFGF within a tissue are probably determined by a com
24#
發(fā)表于 2025-3-25 17:57:41 | 只看該作者
25#
發(fā)表于 2025-3-25 23:06:26 | 只看該作者
Effects of Neurotrophic Factors on the Secretion and Metabolism of the Alzheimer Amyloid Precursor,egenerating and regenerating neuntes. The main component of the plaque amyloid is a small protein (βA4 protein) which derives from a larger amyloid precursor protein (APP). It has been proposed that the neuritic regeneration associated with the neuritic plaques takes place in the presence of a milie
26#
發(fā)表于 2025-3-26 00:11:30 | 只看該作者
27#
發(fā)表于 2025-3-26 07:09:12 | 只看該作者
,The Role of Misdirected Plasticity in Plaque Biogenesis and Alzheimer’s Disease Pathology,or cell loss. This process may represent the plasticity by which the brain can modify its circuitry and compensate for neuronal or synaptic loss after trauma and in a variety of disease states. Lesions of the entorhinal cortex in the rodent brain and degenerative cell loss in this region in Alzheime
28#
發(fā)表于 2025-3-26 10:16:48 | 只看該作者
,Streckenverh?ltnisse. Proportionalit?t, of adult neurons. Administration of neurotrophic factors can attenuate age-related and experimentally induced degeneration and behavioral deficits in animals, suggesting that such molecules may become useful in the treatment of diseases involving structural disintegration of nervous tissue. In part
29#
發(fā)表于 2025-3-26 12:47:35 | 只看該作者
30#
發(fā)表于 2025-3-26 17:41:06 | 只看該作者
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