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Titlebook: Glial Amino Acid Transporters; Arturo Ortega,Arne Schousboe Book 2017 Springer International Publishing AG 2017 Amino Acid Transporters.GA

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21#
發(fā)表于 2025-3-25 07:01:19 | 只看該作者
22#
發(fā)表于 2025-3-25 08:55:30 | 只看該作者
Development of Non-GAT1-Selective Inhibitors: Challenges and Achievements,. Four different transporters have been identified: GAT1, GAT2, GAT3, and the betaine/GABA transporter 1 (BGT1). The GAT1 subtype is the most explored transporter due to its high abundance in the brain and the existence of selective and potent GAT1 inhibitors. Consequently, less is known about the r
23#
發(fā)表于 2025-3-25 14:43:37 | 只看該作者
,Auswahl der Transformatoren?le bei der AEG,n body. However, chronic overexposure to Mn from occupational or environmental sources induces a neurological disorder, characterized by psychiatric, cognitive, and motor abnormalities, referred to as manganism. Mn-induced neurotoxicity is known to target astrocytes since these cells preferentially
24#
發(fā)表于 2025-3-25 15:55:47 | 只看該作者
,Hohe Wechselspannungen und -str?me, also a positive neuromodulator at excitatory glutamate-mediated synapses. Glial cells provide metabolic support to neurons and modulate synaptic activity. Six transporters belonging to three solute carrier families (SLC6, SLC38, and SLC7) are capable of transporting glycine across the glial plasma
25#
發(fā)表于 2025-3-25 22:19:02 | 只看該作者
Grundlagen der Teilentladungsmesstechnik,ly critical in the brain. Taurine participates in osmotic adjustments required to maintain the organization and size of intracellular compartments. It counteracts volume fluctuations in unbalanced transmembrane fluxes of ions and neurotransmitters, preserving the functional synaptic contacts. Taurin
26#
發(fā)表于 2025-3-26 00:11:18 | 只看該作者
27#
發(fā)表于 2025-3-26 04:27:37 | 只看該作者
28#
發(fā)表于 2025-3-26 08:56:50 | 只看該作者
,?l im Zusammenbau mit festen Baustoffen,ely heterogeneous disease in both cause and symptom development, and its mechanisms of pathogenesis remain largely unknown. Excitotoxicity, a process caused by excessive glutamate signaling, is believed to play a substantial role, however. Excessive glutamate release, changes in postsynaptic glutama
29#
發(fā)表于 2025-3-26 13:44:48 | 只看該作者
30#
發(fā)表于 2025-3-26 18:48:05 | 只看該作者
,Der Lichtbogen in Luft und unter ?l,mbrane proteins is the cerebellum. The most abundant glutamatergic synapse in the central nervous system is present in the molecular layer of the cerebellar cortex, and it is entirely wrapped by Bergmann glial cells. The recycling of glutamate involves glutamate and glutamine transporters enriched i
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