Titlebook: Glaucoma Update IV; G. K. Krieglstein (Direktor) Conference proceedings 1991 Springer-Verlag Berlin Heidelberg 1991 Elektrophysiologie.Gla

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Simone Luigi Marasso,Matteo Cocuzzaascular, mechanical, and combined mechanisms have been suggested; however, in spite of a long history of discussion and investigation, an approach to understanding ON damage is not yet clear. Recently, my clinical and experimental investigations have revealed opposing facts to the vascular theory. We will present them in detail.

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,Care of the “High Risk” Newborn Infant,ressive open-angle glaucoma (POAG). We ophthalmologists are, therefore, now requested to establish early diagnosis and effective therapy. To better understanding the mechanism of optic nerve damage in LTG, we need histopathological studies at every stage, but opportunities for such investigations are rare and animal experiments are also difficult.

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TOXIC

The Biology of the Connective Tissue of the Human Optic Nerve Head, nerve head. We have hypothesized that age-related changes and individual differences in the molecular composition of this tissue may underlie the glaucomatous process and contribute to the degeneration of optic nerve fibers. This chapter will review some of the data that we have presented in support of this hypothesis.

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Vascular Reduction Following Axonal Damage in Glaucoma,ascular, mechanical, and combined mechanisms have been suggested; however, in spite of a long history of discussion and investigation, an approach to understanding ON damage is not yet clear. Recently, my clinical and experimental investigations have revealed opposing facts to the vascular theory. We will present them in detail.

Ceramic

The Histopathology of the Optic Nerve in Low-Tension Glaucoma,ressive open-angle glaucoma (POAG). We ophthalmologists are, therefore, now requested to establish early diagnosis and effective therapy. To better understanding the mechanism of optic nerve damage in LTG, we need histopathological studies at every stage, but opportunities for such investigations are rare and animal experiments are also difficult.

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Clinical Features and Systemic Background of Low-Tension Glaucoma,tly below 21 mmHg. Despite the above definition LTG does not always refer to a single clinical entity, but encompasses various etiologic disorders with unknown causes [7]. The pathogenesis of LTG is poorly understood, however, discrimination of cases may be possible from their clinical course; namely, progressive and stationary [5].

Noctambulant

Calibration of a stereo camera system,ing the regulatory response of ocular vessels to physiologic and pharmacologic stimuli. For these studies, we have used a methacrylate intraluminal casting model using a modification of Batson’s compound #17 injected into major arterial vessels, the aorta or carotid arteries of experimental animals

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Klaus Linkwitz,Ulrich Hangleiter of progress is surprising in view of the facts that nearly 100 years ago the major catecholamine component of the adrenal gland was isolated and named epinephrine [1], and that adrenal gland extracts were shown to lower IOP when injected subconjunctivally [2] or applied topically in other early exp

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