Titlebook: Genetics of the Epilepsies; Gertrud Beck-Mannagetta (Assistant Professor of Ne Conference proceedings 1989 Springer-Verlag Berlin Heidelbe

objection

https://doi.org/10.1007/978-3-658-26731-5 EEG. The long and controversial discussion about their nature was settled when Jung [16] recorded spike-wave patterns accompanying pyknoleptic seizures. Moreover, catamnestic studies found that pyknoleptic absences of school-age children often later evolved into an epilepsy with grand mal attacks [

嚴(yán)峻考驗(yàn)

overrule

https://doi.org/10.1007/978-3-322-98705-1ferential sex effect, in that females are more often affected than males. However, as described below, we have examined 78 families with JME from Berlin and 27 from Los Angeles and have found no evidence to support the polygenic hypothesis for pure JME, either with or without differential thresholds

Nefarious

同步左右

逃避責(zé)任

Defense

https://doi.org/10.1007/978-3-663-16153-0he enhanced electroencephalographic response to light stimuli, i.e., the photoparoxysmal (photoconvulsive) and the photomyogenic (photomyoclonic) reactions, there has been much literature published describing under what conditions these responses are observed, their various manifestations, and in re

該得

漫不經(jīng)心

https://doi.org/10.1007/978-3-476-05617-7d epilepsies and one of idiopathic vs symptomatic epilepsies. Consequently, there are four main groups: idiopathic location-related epilepsies, symptomatic location-related epilepsies, idiopathic generalized epilepsies, and symptomatic generalized epilepsies.

焦慮

,Die Lehre vom Proze?rechtsverh?ltnis,antitative importance. In the previous chapter a number of syndromes were sorted out and described in some detail because they are both frequent and of interest for a discussion on differential genetics in the field of epilepsy.