Titlebook: Epstein Barr Virus Volume 1; One Herpes Virus: Ma Christian Münz Book 2015 The Editor(s) (if applicable) and The Author(s), under exclusive

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https://doi.org/10.1007/978-3-642-91525-3geographic variation of EBV strains independent of the type 1/type 2 classification and systematic investigation of the relationship between viral strains, infection and disease are now becoming possible. So we should consider more directly whether viral sequence variation might play a role in the incidence of some EBV-associated diseases.

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EBV-Specific Immune Response: Early Research and Personal Reminiscencesefinition of the humoral response to a variety of EBV proteins dominated the early literature and was instrumental in providing the key evidence for the association of the virus with infectious mononucleosis (IM), Burkitt’s lymphoma (BL), and nasopharyngeal carcinoma (NPC). Each of these disease ass

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Epstein–Barr Virus Strain Variationrogress in DNA sequencing has resulted in many new Epstein–Barr virus (EBV) genome sequences becoming available. EBV isolates worldwide can be grouped into type 1 and type 2, a classification based on the EBNA2 gene sequence. Type 1 transforms human B cells into lymphoblastoid cell lines much more e

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Chromatin Structure of Epstein–Barr Virus Latent Episomesnt tissue types with remarkable variation and plasticity in viral transcription and replication. During latency, the viral genome persists as a multi-copy episome, a non-integrated-closed circular DNA with nucleosome structure similar to cellular chromosomes. Chromatin assembly and histone modificat

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The Epigenetic Life Cycle of Epstein–Barr Virust infection, which is the prominent hallmark of this member of the herpesvirus family. EBV has become an important model for many aspects of herpesviral latency, but the molecular steps and mechanisms that lead to and promote viral latency have only emerged recently. It now appears that the virus ex

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Epstein–Barr Virus: From the Detection of Sequence Polymorphisms to the Recognition of Viral Typeseous geographic distribution across the world. The virus possesses a 170-kb-large genome that encodes for multiple proteins and non-coding RNAs. Early on there have been numerous attempts to link particular diseases with particular EBV strains, or at least with viral genetic polymorphisms. This has

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