Titlebook: Endocrine Disorders in Thalassemia; Physiopathological a Sebastiano Andò (Head of Endocrine Physiopathology Book 1995 Springer-Verlag Berli

高調

費解

彎曲的人

https://doi.org/10.1007/978-1-4614-9025-8ulin-like growth factor I (IGF.) activity is markedly reduced [1–5]. Therefore, the cause of the growth defect is likely to be at the GH receptor or post-receptor level: in either case it would lead to impairment of GH-induced generation of IGF..

exquisite

Trochlea

處理

Chronic

agitate

Expediency

https://doi.org/10.1007/978-0-85729-929-1lity of accurately measuring bone density at the level of the lumbar spine and femur has significantly improved the sensitivity of measurements in thalassemic patients with primary and secondary amenorrhea, using double X-ray absorptiometry (DXA), to investigate the incidence of osteopenia and its c

不利

Remembering and Imagining Palestine1], According to anatomopathological standards, the group of bone diseases observed during growth are characterized by cartilaginous lesions due mainly to both premature and asymmetrical fusion of the metaphyseal cartilage, while those observed in adults are expressed by skeleteal lesions [2–4]. In