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Titlebook: Diagnosis and Management of Muscle Disease; Albert P. Galdi Book 1984 Spectrum Publications, Inc. 1984 muscle

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發(fā)表于 2025-3-21 18:34:47 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Diagnosis and Management of Muscle Disease
編輯Albert P. Galdi
視頻videohttp://file.papertrans.cn/271/270574/270574.mp4
叢書(shū)名稱(chēng)Neurologic Illness
圖書(shū)封面Titlebook: Diagnosis and Management of Muscle Disease;  Albert P. Galdi Book 1984 Spectrum Publications, Inc. 1984 muscle
描述Neurologists and non-neurologists alike can no longer ignore diseases of the neuromuscular system. The old dogma that these disorders are both uncommon and untreatable has lost its validity. Recent technological advances have enabled us to study more precisely muscle and nerve anatomy, physiology and biochem- istry. Because of this progress, we are now recognizing new neuromuscular di- seases as well as diagnosing more subtle cases of myasthenia gravis, myotonia, and metabolic myopathies. Treatment of the neuromuscular diseases has also un- dergone dramatic change based on new discoveries in the fields of immunology and pharmacology. No longer are myotonia, periodic paralysis, and malignant hyperthermia untreatable medical curiosities. No longer are cases of steroid-un- responsive myositis given up as hopeless. Because of all these advances, non-sur- gical physicians and especially neurologists must update their knowledge regard- ing the neuromuscular disorders. This book, it is hoped, will help such clinicians in dealing with this task. Emphasis has been placed on the diagnosis and management of these disorders rather than on their pathophysiology. The more uncommon diseases and t
出版日期Book 1984
關(guān)鍵詞muscle
版次1
doihttps://doi.org/10.1007/978-94-011-6335-4
isbn_softcover978-94-011-6337-8
isbn_ebook978-94-011-6335-4
copyrightSpectrum Publications, Inc. 1984
The information of publication is updating

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沙發(fā)
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Irene Tiemann-Boege,Andrea Betancourtquired for this process, and the phosphate bonds of adenosine triphosphate (ATP) provide the immediate source of this energy. Hydrolysis of the terminal phosphate bond of one mole of ATP liberates 7 to 8 kcal of energy.
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PacBio for Haplotyping in Gene FamiliesUntil recently, it was not widely appreciated that muscle disease occurs in association with alcoholism. However, as early as the middle of the nineteenth century, clinicians realized that neurogenic disease alone could not explain all the motor symptoms manifested by their alcoholic patients.
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Disorders of Muscle Energy Metabolism and Mitochondria,quired for this process, and the phosphate bonds of adenosine triphosphate (ATP) provide the immediate source of this energy. Hydrolysis of the terminal phosphate bond of one mole of ATP liberates 7 to 8 kcal of energy.
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Ultrastructure of Pollen Embryogenesis mastered. The neuromuscular junction (Figure 3-1) consists of a motor nerve ending (presynaptic terminal) and a postsynaptic muscle fiber membrane separated by a space, the synaptic cleft. The motor nerve terminal is filled with packets called synaptic vesicles, which contain acetycholine (ACh), th
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