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Titlebook: Diabetes and Cardiovascular Disease; Michael T. Johnstone,Aristidis Veves Book 20011st edition Springer Science+Business Media New York 20

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樓主: Grievous
21#
發(fā)表于 2025-3-25 04:18:41 | 只看該作者
22#
發(fā)表于 2025-3-25 11:27:25 | 只看該作者
Effects of Insulin on the Vascular System To accomplish this task for the ever changing tissue requirements without compromising the blood supply of vital organs, the vascular system responds in a variety of ways. It responds at the local tissue level via the release of short-acting vasoactive hormones, which redirect blood flow from less
23#
發(fā)表于 2025-3-25 14:49:08 | 只看該作者
Effect of Diabetes on Endothelial Function-and macrovascular tissues. After the discovery of insulin in 1921, the major cause of morbidity and mortality in diabetic patients became cardiovascular complications . The incidence of coronary artery disease (CAD) in patients with diabetes or insulin resistance syndrome is increased in diabetic p
24#
發(fā)表于 2025-3-25 16:52:38 | 只看該作者
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發(fā)表于 2025-3-25 23:26:39 | 只看該作者
26#
發(fā)表于 2025-3-26 03:19:28 | 只看該作者
The Renin-Angiotensin System in Diabetic Cardiovascular Complicationsomeostasis. Reports from a growing number of clinical trials have demonstrated that suppression of the RAS by angiotensin-converting enzyme (ACE) inhibitors reduces the onset and/or progression of certain renal ., retinal ., and cardiovascular . complications of diabetes mellitus. These findings, al
27#
發(fā)表于 2025-3-26 08:04:37 | 只看該作者
28#
發(fā)表于 2025-3-26 12:02:44 | 只看該作者
Diabetes and Dyslipidemiadisease (CHD) . The increase in risk is particularly evident in both younger age groups and in women. Women with type 2 diabetes appear to lose a great deal of the protection that characterizes nondiabetic females. Furthermore, patients with diabetes have a 50% greater in-hospital mortality and a tw
29#
發(fā)表于 2025-3-26 13:45:19 | 只看該作者
30#
發(fā)表于 2025-3-26 20:03:21 | 只看該作者
Diabetes and Atherosclerosisns result from the narrowing and thrombosis of coronary, cerebral, and peripheral vessels. The study of factors that may uniquely contribute to the accelerated development of atherosclerosis in diabetes has been an ongoing process for several years. The concepts behind both the pathogenic mechanisms
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