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Titlebook: Diabetes Epidemiology, Genetics, Pathogenesis, Diagnosis, Prevention, and Treatment; Enzo Bonora,Ralph A. DeFronzo Reference work 2018 Spr

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發(fā)表于 2025-3-28 17:22:10 | 只看該作者
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發(fā)表于 2025-3-28 20:50:29 | 只看該作者
https://doi.org/10.1007/978-3-030-25759-0factors, and morbidity/mortality in diabetic patients..Three main topics are included in this chapter: (1) the effects of dietary treatment on body weight control in diabetic patients; (2) the optimal dietary composition in order to achieve blood glucose control and reduce other cardiovascular risk
43#
發(fā)表于 2025-3-29 00:17:34 | 只看該作者
David Renz,Lonneke Roza,Frans-Joseph Simonsal metabolic diseases, including diabetes mellitus. The non-pharmacological low-cost nature, along with the health-related benefits, increases the therapeutical . of regular physical activity. In the comprehensive approach of diabetes management, regular physical activity reduces risk of many diseas
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發(fā)表于 2025-3-29 05:48:42 | 只看該作者
https://doi.org/10.1007/978-3-030-25759-0es. In the meantime, better understanding of the pathophysiology of hyperglycemia in Type 2 diabetes has been gained and the identification of several pathogenitic mechanisms has enabled moving from serendipitous discovery – as for sulfonylureas and metformin – to the development of agents with more
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發(fā)表于 2025-3-29 08:44:35 | 只看該作者
Diabetes Epidemiology, Genetics, Pathogenesis, Diagnosis, Prevention, and Treatment978-3-319-45015-5Series ISSN 2510-1927 Series E-ISSN 2510-1935
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發(fā)表于 2025-3-29 14:02:57 | 只看該作者
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發(fā)表于 2025-3-29 19:48:30 | 只看該作者
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發(fā)表于 2025-3-30 02:25:29 | 只看該作者
Diagnostic Criteria and Classification,l estimates were published in 2000. Nevertheless, diabetes prevalence estimates are highly dependent on factors such as data sources and quality, method used to diagnose diabetes, diagnostic criteria, and modelling assumptions. This chapter includes a review of the development of the current diagnos
50#
發(fā)表于 2025-3-30 04:16:56 | 只看該作者
Epidemiology and Risk Factors of Type 1 Diabetes,ta cells in the pancreas. Genetic, epigenetic, metabolic, and environmental factors act together to precipitate the onset of the disease. Clinical T1D represents the end stage of a process resulting from the progressive beta-cell destruction following an asymptomatic period that may last for years.
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