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Titlebook: DNA Repair in Cancer Therapy; Lawrence C. Panasci,Moulay A. Alaoui-Jamali Book 2004 Springer Science+Business Media New York 2004 BRCA.DNA

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樓主: calcification
41#
發(fā)表于 2025-3-28 18:01:38 | 只看該作者
42#
發(fā)表于 2025-3-28 20:11:11 | 只看該作者
43#
發(fā)表于 2025-3-29 00:15:03 | 只看該作者
44#
發(fā)表于 2025-3-29 05:12:46 | 只看該作者
Tacit Knowledge, Rule-following and Learningngements, termed V(D)J recombination, the only known form of site-specific DNA rearrangement in vertebrates. This assembly process is highly conserved throughout evolution and present in most of the vertebrates analyzed so far.
45#
發(fā)表于 2025-3-29 10:31:35 | 只看該作者
https://doi.org/10.1007/978-1-4471-1729-2s that the molecules under normal circumstances in their wild-type state could prove to be targets of modern molecular chemotherapy. Ironically, the very molecular basis of this hypersensitivity can, in the end, be a cause of acquisition of clinical resistance.
46#
發(fā)表于 2025-3-29 14:45:05 | 只看該作者
47#
發(fā)表于 2025-3-29 16:45:01 | 只看該作者
V(D)J Recombination and DNA Double-Strand-Break Repair,ngements, termed V(D)J recombination, the only known form of site-specific DNA rearrangement in vertebrates. This assembly process is highly conserved throughout evolution and present in most of the vertebrates analyzed so far.
48#
發(fā)表于 2025-3-29 23:02:50 | 只看該作者
Inherited Disorders of Genomic Instability and Cancer Susceptibility,s that the molecules under normal circumstances in their wild-type state could prove to be targets of modern molecular chemotherapy. Ironically, the very molecular basis of this hypersensitivity can, in the end, be a cause of acquisition of clinical resistance.
49#
發(fā)表于 2025-3-30 02:10:53 | 只看該作者
50#
發(fā)表于 2025-3-30 07:22:24 | 只看該作者
Book 2004ncologists dealing with hallmark "relapse" or "drug resistance" phenomena on a daily basis-the many exciting new uses of DNA repair inhibitors, either alone or in combination with anticancer therapies.
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