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Titlebook: DNA Repair and Mutagenesis in Eukaryotes; W. M. Generoso,M. D. Shelby,F. J. Serres Book 1980 Springer Science+Business Media New York 1980

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11#
發(fā)表于 2025-3-23 09:42:29 | 只看該作者
Articular Cartilage of the Knee lethals depend on a repair system which in a number of genetically nonrelated strains shows different repair capacities. Sister chromatid exchanges (SCE) are easily studied as ring chromosome losses. They develop through a genotype controlled mechanism from premutational lesions. Stocks with partic
12#
發(fā)表于 2025-3-23 17:48:33 | 只看該作者
13#
發(fā)表于 2025-3-23 21:18:00 | 只看該作者
14#
發(fā)表于 2025-3-24 01:45:53 | 只看該作者
Articulating Novelty in Science and Artse, contact-inhibited mouse cells. During the first hour of repair, approximately 90% of x-ray induced single strand breaks were rejoined whereas most of the remaining breaks were rejoined more slowly during the next 5 hr. The number of residual non-rejoined single strand breaks was approximately pr
15#
發(fā)表于 2025-3-24 06:17:17 | 只看該作者
16#
發(fā)表于 2025-3-24 07:55:57 | 只看該作者
Articular Cartilage of the Kneeion repair are described in terms of mechanisms that yield patch sizes of 0, 1, a few (short patch), and many (long patch) bases. The biological and molecular ways of measuring the effects of replication on a damaged template are presented, as are various models of postreplication repair.
17#
發(fā)表于 2025-3-24 12:18:09 | 只看該作者
18#
發(fā)表于 2025-3-24 15:45:28 | 只看該作者
19#
發(fā)表于 2025-3-24 22:11:07 | 只看該作者
https://doi.org/10.1007/978-1-4684-3842-0DNA; cells; chromosome; mutagen; mutagenesis; mutation; production
20#
發(fā)表于 2025-3-25 00:16:45 | 只看該作者
978-1-4684-3844-4Springer Science+Business Media New York 1980
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