Titlebook: Cytokines and Joint Injury; Wim B. Berg,Pierre Miossec Book 2004 Springer Basel AG 2004 Arthritis.bone.cytokine.cytokines.diseases.inflamm

頭盔

Cell-cell contact in chronic inflammation: the importance to cytokine regulation in tissue destructnd direct cellular interactions [.-.]. In pathologic conditions, the production of cytokines and MMP by infiltrating and resident tissue cells escapes regulatory mechanisms. The activity of proinflammatory cytokines is counterbalanced by numerous mechanisms of which cytokine inhibitors — IL-1 recept

騎師

The role of proteinases in joint tissue destruction,ng growth and development, synthesis of matrix components exceeds the rate of degradation; reduction in the rate of matrix synthesis and an increase in the rate of degradation occur during matrix resorption [.] .

責(zé)怪

Postmenopause

defuse

拋射物

The IL-1 family: The role of IL-1 and IL-18 in inflammation, inflammation and autoimmune diseases. The most salient and relevant properties of IL-1 in inflammation are the initiation of cyclooxygenase type 2 (COX-2), type 2 phospholipase A and inducible nitric oxide syntha se (iNOS). This accounts for the large amount of prostaglandin-E. (PGE.), platelet act

Bother

憤憤不平

Is IL-6 a therapeutic target?, the multiple joints [.]. The pathological features of the affected joints consist of hyperplasia of inflamed synovium and erosion of cartilage and bone where the pannus tissue invades. There are numerous immunocompetent cells such as T cells, macrophages, dendritic cells and plasma cells infiltrati

etidronate

The clinical use of anti-cytokine therapy in rheumatic diseases,. Although the precise aetiopathogenesis of these inflammatory arthropathies have not been elucidated, inflammation and damage are common features. Active joint inflammation results in pain, stiffness and dysfunction. Chronic synovitis leads to cartilage and bone damage through mechanisms that have

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