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Titlebook: Copper and Zinc in Inflammatory and Degenerative Diseases; K. D. Rainsford,R. Milanino,G. P. Velo Book 1998 Springer Science+Business Medi

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書目名稱Copper and Zinc in Inflammatory and Degenerative Diseases
編輯K. D. Rainsford,R. Milanino,G. P. Velo
視頻videohttp://file.papertrans.cn/239/238174/238174.mp4
圖書封面Titlebook: Copper and Zinc in Inflammatory and Degenerative Diseases;  K. D. Rainsford,R. Milanino,G. P. Velo Book 1998 Springer Science+Business Medi
描述Abnormal metabolism and distribution of both copper and zincoccurs in many inflammatory and degenerative diseases. The pattern ofthese changes varies at different stages of these diseases and withdiffering types of conditions. The corollary to this situation is thepossibility of using drugs or metal complexes to modify both theperturbed status of copper and zinc and thus the disease states. .This book comprehensively reviews the clinical and experimental dataon the changes in copper and zinc status in different diseases and theuse of various complexes of these metals or drugs to treat a diversityof inflammatory and degenerative conditions.
出版日期Book 1998
關(guān)鍵詞autoimmune disease; autoimmune diseases; cancer; chemistry; diseases; distribution; inflammation; metabolis
版次1
doihttps://doi.org/10.1007/978-94-011-3963-2
isbn_softcover978-94-010-5757-8
isbn_ebook978-94-011-3963-2
copyrightSpringer Science+Business Media Dordrecht 1998
The information of publication is updating

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CNH: The Ongoing Challenges of Change. Relevant to the treatment of the rheumatic diseases, zinc deficiency profoundly disturbs the immune function and the host defense mechanism, resulting in lymphoid atrophy, impaired delayed hypersensitivity and T-helper cell dysfunction [2–5]
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Zinc and copper in the treatment of rheumatic diseases,. Relevant to the treatment of the rheumatic diseases, zinc deficiency profoundly disturbs the immune function and the host defense mechanism, resulting in lymphoid atrophy, impaired delayed hypersensitivity and T-helper cell dysfunction [2–5]
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https://doi.org/10.1007/978-1-4020-8948-0rescription of chemical therapeutics in medicine (Figure 1). Paracelsus sharply broke with traditional beliefs when he applied his interest in alchemy and metals to the treatment of his patients. In so doing, he earned himself the title ‘father of pharmacology’ [1]. Zinc sulphate (sal vitrioli) was
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https://doi.org/10.1007/978-1-4020-8948-0ectron transfer reactions. In-vivo oxygen chemistry is dependent on and regulated by copper proteins: all reduction states of dioxygen are connected with copper proteins and related copper compounds (Figure 1).
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