Titlebook: Chronic Pancreatitis; Research and Clinica H. G. Beger,M. Büchler,P. Malfertheiner Conference proceedings 1990 Springer-Verlag Berlin Heide

aggression

書目名稱Chronic Pancreatitis影響因子(影響力)




書目名稱Chronic Pancreatitis影響因子(影響力)學(xué)科排名




書目名稱Chronic Pancreatitis網(wǎng)絡(luò)公開度




書目名稱Chronic Pancreatitis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Chronic Pancreatitis被引頻次




書目名稱Chronic Pancreatitis被引頻次學(xué)科排名




書目名稱Chronic Pancreatitis年度引用




書目名稱Chronic Pancreatitis年度引用學(xué)科排名




書目名稱Chronic Pancreatitis讀者反饋




書目名稱Chronic Pancreatitis讀者反饋學(xué)科排名

Fillet,Filet

Incidence and Prevalence of Chronic Pancreatitis] and 5% [2]. The clinical picture was only sporadically mentioned in the medical literature up to 1946, when Comfort et al. [3] presented the first detailed clinical material of chronic pancreatitis, comprising 29 cases. Since that time, studies on etiology, clinical picture, medical and surgical t

主動(dòng)

Role of Nutrition in Chronic Pancreatitisical stimuli, the most prominent of which are nutritional in origin. Normal pancreatic function and structure are altered by a number of nutritional factors which include changes in intake of major dietary components (proteins, carbohydrates, and fats) as well as vitamins and other micronutrients an

Dorsal-Kyphosis

Sad570

Hyperplasia

Hyperplasia

Focal Necrosis: Primary Event in the Pathogenesis of Chronic Pancreatitis?20]. Yet its significance for the pathogenesis of chronic pancreatitis has been neglected for many years, owing to the widely accepted view that acute and chronic pancreatitis differ in etiology and pathogenesis [18]. Here we will briefly review the pathology of focal necrosis and pseudocysts in chr

細(xì)節(jié)

Intersect

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