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Titlebook: Chronic Inflammation; Mechanisms and Regul Masayuki Miyasaka,Kiyoshi Takatsu Book 2016 Springer Japan 2016 Chronic diseases.Proinflammatory

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樓主: LH941
51#
發(fā)表于 2025-3-30 11:47:05 | 只看該作者
Conditions Favouring Political Theoryenic signals, and it was initially identified as a tumour-suppression mechanism. Recent studies, however, revealed that senescent cells have the potential to secrete numerous inflammatory cytokines, chemokines, growth factors, and matrix-remodeling factors, because unlike apoptotic cells, senescent
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發(fā)表于 2025-3-30 12:47:31 | 只看該作者
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發(fā)表于 2025-3-30 17:53:52 | 只看該作者
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發(fā)表于 2025-3-31 00:33:03 | 只看該作者
Masayuki Miyasaka,Kiyoshi TakatsuProvides the most up-to-date summary of cutting-edge research on chronic inflammation.Clarifies the molecular link between chronic inflammation and various diseases including cancer, cardiovascular di
55#
發(fā)表于 2025-3-31 01:24:06 | 只看該作者
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發(fā)表于 2025-3-31 06:33:09 | 只看該作者
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發(fā)表于 2025-3-31 12:34:39 | 只看該作者
Cellular and Molecular Mechanisms of Chronic Inflammation-Associated Organ FibrosisECM deposition in injured tissues is a natural part of the wound healing process that facilitates efficient restoration of tissue integrity. However, if the injury persists, the excessive accumulation of ECM leads to the loss of organ function and eventual organ failure. Persistent injuries arise du
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發(fā)表于 2025-3-31 14:35:39 | 只看該作者
Sema4A and Chronic Inflammationcates that several semaphorins, so-called ‘immune semaphorins’, have important functions in various phases of pathological and physiological immune responses, such as cell–cell interactions, immune-cell activation, differentiation, and trafficking/migration. For example, Sema4A, a class IV transmemb
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發(fā)表于 2025-3-31 17:35:34 | 只看該作者
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發(fā)表于 2025-3-31 23:35:36 | 只看該作者
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