Titlebook: Chlamydia pneumoniae and Chronic Diseases; Proceedings of the S Johanna L’age-Stehr Conference proceedings 2000 Springer-Verlag Berlin Heid

Critical

State of the art in the diagnosis of acute and chronic , infectionsted that there are differences in the antibody response to primary infection, reinfection and reactivated infection, there are limited data correlating serology with culture. This is further complicated by the lack of standardized methods for serology and nonculture methods such as polymerase chain reaction (PCR).

流眼淚

Chlamydia infections and arthritisday presents the data on Alzheimer disease. A very similar clinical picture can be seen after gut infections with enterobacteria, and some of the data I will present for . also include data from .- and .-induced reactive arthritis.

嚴(yán)重傷害

Pathogenic mechanisms of chronic ,nd the serotypes L1, L2, and L3, responsible for the lymphogranuloma venereum. . is not divided into serotypes so far. It causes respiratory infections, chronic juvenile arthritis and is discussed in relation to the pathogenesis of intrinsic asthma and atherosclerosis.

Integrate

Role of , in severe asthma and COPD: Epidemiology and treatmentximately 10 % of asthma exacerbations [3,6]. Most studies described patients with mild to moderate exacerbations whereas arecent study by our group, while confirming the role of this pathogen in mild to moderate asthma exacerbations, suggests that . must also be considered as a cause of severe asthma attacks [7] (Table 1).

rheumatology

Why antibiotics against , for treatment of atherosclerotic disease could fail in PCR or immunocytochemistry, then PCR alone detects only 50 % of all the patients having . and immunocytochemistry detects over 80%. Usually PCR is considered a much more sensitive technique than immunocytochemistry. So why is PCR less sensitive than immunocytochemistry?

atrophy

Harpoon

綠州

Type-1-Diabetes

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