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Titlebook: Chirurgisches Forum 2002; für experimentelle u J. R. Siewert,E. Neugebauer,M. D. Menger Conference proceedings 2002 Springer-Verlag Berlin

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31#
發(fā)表于 2025-3-26 21:59:41 | 只看該作者
Yasuyuki Nagasawa,Yoshihiko Kannoased CD95 cell surface staining and, in parallel, sensitivity to CD95-induced apoptosis. These data suggest that PKCμ may exert its anti-apoptotic function, at least in part, by reducing the fraction of CD95 on the cell surface.
32#
發(fā)表于 2025-3-27 03:23:56 | 只看該作者
Joaquim A. O. Barros,Liberato Ferraragrading..We present the first report on frequent occurrence of mutations in the mtDNA D-loop regions in adenocarcinomas in Barrett’s esophagus. Furthermore, this study strongly supports the hypothesis that oxidative damage is an important mechanism for the induction of adenocarcinoma in Barrett’s esophagus.
33#
發(fā)表于 2025-3-27 08:19:44 | 只看該作者
34#
發(fā)表于 2025-3-27 10:47:01 | 只看該作者
Resistenz gegenüber CD95-vermittelter Apoptose im duktalen Pankreasadenokarzinom: Die Rolle der Protased CD95 cell surface staining and, in parallel, sensitivity to CD95-induced apoptosis. These data suggest that PKCμ may exert its anti-apoptotic function, at least in part, by reducing the fraction of CD95 on the cell surface.
35#
發(fā)表于 2025-3-27 14:59:02 | 只看該作者
Hohe Mutationsfrequenz in der mitochondrialen DNA D-Loop Region bei Adenocarcinomen im Barrett-?sophgrading..We present the first report on frequent occurrence of mutations in the mtDNA D-loop regions in adenocarcinomas in Barrett’s esophagus. Furthermore, this study strongly supports the hypothesis that oxidative damage is an important mechanism for the induction of adenocarcinoma in Barrett’s esophagus.
36#
發(fā)表于 2025-3-27 20:50:34 | 只看該作者
K. Tanaka,N. Nitta,K. Akita,T. Watanabeas inhibited by antisense oligonucleotides against VEGF-RII, but not by those against VEGF-RI. Therefore VEGF represents an autocrine growth factor in human pancreatic cancer. VEGF-RII appears to be a key protein in angiogenesis and mitogenesis and its blockade offers a new therapeutic target.
37#
發(fā)表于 2025-3-28 00:07:07 | 只看該作者
38#
發(fā)表于 2025-3-28 04:07:36 | 只看該作者
VEGF-RII nicht aber VEGF-RI Expression korreliert mit Tumordifferenzierung, Angiogenese und Tumorzelas inhibited by antisense oligonucleotides against VEGF-RII, but not by those against VEGF-RI. Therefore VEGF represents an autocrine growth factor in human pancreatic cancer. VEGF-RII appears to be a key protein in angiogenesis and mitogenesis and its blockade offers a new therapeutic target.
39#
發(fā)表于 2025-3-28 09:22:33 | 只看該作者
40#
發(fā)表于 2025-3-28 12:34:57 | 只看該作者
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