Titlebook: Chest Pain with Normal Coronary Angiograms: Pathogenesis, Diagnosis and Management; Juan Carlos Kaski (BHF Sugden Reader in Clinical C Boo

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R?ntgen-Nativdiagnostik und Tomographieal ischemia induced by pacing in the absence of detectable left ventricular dysfunction in a group of patients with angina and normal coronary angiograms [.]. This term proved to be well chosen as during the last quarter of the century this syndrome continued to be the object of conflicting reports and a variety of pathogenetic hypotheses.

強(qiáng)化

https://doi.org/10.1007/978-3-642-69557-5aphy provides merely a luminogram of the artery. This is exemplified by the demonstration of fibrous or lipid containing plaques within the artery wall using intracoronary ultrasound and Doppler, which may escape detection by contrast arteriography, particularly when located at the coronary ostium [.].

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Springer Series in Computational Mathematics, accounts of similar patients can be found in the literature up to a century earlier. Most early descriptions emphasize organic abnormalities in explaining the symptoms: but there has been an increasing recognition that psychological abnormalities are also common. Several recent studies, reviewed b

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Guidelines for the Use of QUADPACK,n [.–.], to the extent that chest pain of uncertain origin has now become a rare indication for esophageal manometry in the United States [.]. The reasons for this include changing perceptions about the relevance of the commonest manometric diagnosis, the so called “nutcracker esophagus” [.,.,.], th

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Quantentheorie atomarer Systeme,out any specific cardiac, esophageal, musculo-skeletal, or psychiatric disorders. There are many clues indicating that this anginal syndrome is in fact many different syndromes giving rise to a similar symptomatology. However, a general abnormality of nociception could incorporate many of the result

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https://doi.org/10.1007/978-3-642-65289-9t pain, ischemic-like changes on the stress ECG yet a normal coronary arteriogram). In this chapter, we review advances in knowledge of the pathophysiology of this condition which have been obtained by means of positron emission tomography (PET). The context in which the PET studies have been perfor