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Titlebook: Cerebral Circulation and Metabolism; Sixth International Thomas W. Langfitt,Lawrence C. McHenry,Harry Wollm Book 1975 Springer-Verlag New

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發(fā)表于 2025-3-23 11:44:37 | 只看該作者
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發(fā)表于 2025-3-23 13:57:23 | 只看該作者
The Effect of Lowered Cardiac Output on Cerebral Blood FlowThe purpose of the present study is to investigate the effect of a simultaneous lowering of cardiac output (CO) and mean systemic arterial pressure (MAP) by ventricular pacing on cerebral blood flow (CBF) in the dog; i.e., to determine if cerebral autoregulation is adequate to maintain CBF in the presence of a concomitant fall in both MAP and CO.
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發(fā)表于 2025-3-23 18:51:10 | 只看該作者
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發(fā)表于 2025-3-24 01:24:57 | 只看該作者
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發(fā)表于 2025-3-24 06:33:44 | 只看該作者
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發(fā)表于 2025-3-24 13:13:25 | 只看該作者
Book 1975ach session. The last session con- Blood Flow held in Philadelphia on June 6-9, tains discussions of several topics which were felt to be of sufficient importance and interest to 1973. Since the first meeting on cerebral blood flow, held in Lund in 1964, the number of par- warrant extended considera
18#
發(fā)表于 2025-3-24 16:01:19 | 只看該作者
irman of each session. The last session con- Blood Flow held in Philadelphia on June 6-9, tains discussions of several topics which were felt to be of sufficient importance and interest to 1973. Since the first meeting on cerebral blood flow, held in Lund in 1964, the number of par- warrant extended
19#
發(fā)表于 2025-3-24 20:29:59 | 只看該作者
Mund-Kiefer-Gesichts-Chirurgie (MKG),onstriction of precapillary vessels and is apparently independent of intrinsic or extrinsic neural control. The mechanism of this response is apparently myogenic (12), and it is thought to function physiologically to help maintain capillary hydrostatic pressure within normal limits during fluctuation in venous pressure (6, 12).
20#
發(fā)表于 2025-3-25 02:03:16 | 只看該作者
Effects of Local Increases of Venous Pressure on Canine Cerebral Hemodynamicsonstriction of precapillary vessels and is apparently independent of intrinsic or extrinsic neural control. The mechanism of this response is apparently myogenic (12), and it is thought to function physiologically to help maintain capillary hydrostatic pressure within normal limits during fluctuation in venous pressure (6, 12).
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