Titlebook: Cellular Interactions in Cardiac Pathophysiology; Ján Slezák,Attila Ziegelh?ffer Book 1995 Springer Science+Business Media Dordrecht 1995

JOT

Oxidative stress and heart failureerve and increased oxidative stress. The latter may have a causal role as suggested by the protection seen with antioxidant treatment in acute as well as in chronic heart failure. It is becoming increasingly apparent that, anytime the available antioxidant reserve in the cell becomes inadequate, myo

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maculated

Angiotensin converting enzyme inhibitors, left ventricular hypertrophy and fibrosisin blood pressure or on plasma ACE activity but also prevented LVH after aortic banding. The antihypertrophic effect of the higher and lower doses of ramipril, as well as the antihypertensive action of the higher dose of ramipril, was abolished by coadministration of the kinin receptor antagonist ic

LAVA

JADED

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landmark

Intersect

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