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Titlebook: Cardiovascular Neuroendocrinology; Gina L. C. Yosten,J. Thomas Cunningham Book 2023 The Editor(s) (if applicable) and The Author(s), under

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發(fā)表于 2025-3-25 06:59:17 | 只看該作者
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發(fā)表于 2025-3-25 10:04:27 | 只看該作者
Central Neuroendocrine Control of the Cardiovascular Systemressure are exerted through modulation and integration of its neuroendocrine and autonomic functionally segregated parts. Neuroendocrine PVN activity is conveyed by magnocellular neurons (MCNs) and some portion of parvocellular neurons (PCNs). MCNs are the main source of vasopressin (VP)?and oxytoci
23#
發(fā)表于 2025-3-25 11:40:05 | 只看該作者
Neuroendocrine Control of the Vascular Systemides involved in cardiovascular regulation. These hormones and peptides, acting on specific receptors in different organs and tissues of the cardiovascular system, regulate vasodilation and vasoconstriction of the vessels, chronotropic and ionotropic effects in the heart, and water and electrolyte h
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發(fā)表于 2025-3-25 19:03:26 | 只看該作者
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發(fā)表于 2025-3-25 22:17:40 | 只看該作者
Cardiovascular Neuroendocrinology of Pregnancyological processes related to growth, metabolism, fluid homeostasis, and sexual development. During pregnancy, the placenta is integrated into the complex feedback systems among the hypothalamus, pituitary, and its target organs and contributes to the maintenance of a supportive maternal–fetal envir
26#
發(fā)表于 2025-3-26 01:29:26 | 只看該作者
Cerebrovascular Function in Agingunction becomes an increasing priority. In this chapter, we highlight the recent advances in our understanding of cerebrovascular function with aging by covering work in animals and humans. Advancing age impacts cerebrovascular function through several mechanisms, such as changes in vascular functio
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發(fā)表于 2025-3-26 08:01:25 | 只看該作者
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發(fā)表于 2025-3-26 09:14:44 | 只看該作者
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發(fā)表于 2025-3-26 13:23:44 | 只看該作者
30#
發(fā)表于 2025-3-26 16:50:04 | 只看該作者
https://doi.org/10.1007/978-3-642-82225-4veral models of hypertension, increases excitation from extrinsic osmoreceptors. Together, these observations suggest that changes in osmosensation may contribute to alterations in AVP release involved in the pathogenesis of salt-sensitive hypertension.
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