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Titlebook: Cardio-Renal Clinical Challenges; David Goldsmith,Adrian Covic,Jonas Spaak Book 2015 Springer International Publishing Switzerland 2015 An

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發(fā)表于 2025-3-21 17:35:07 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Cardio-Renal Clinical Challenges
編輯David Goldsmith,Adrian Covic,Jonas Spaak
視頻videohttp://file.papertrans.cn/222/221855/221855.mp4
概述Discusses the evidence for best management of chronic kidney disease (CKD) in the context of cardiorenal syndrome (CRS), as well as the reasons for the complex interplay between the cardiac and renal
圖書(shū)封面Titlebook: Cardio-Renal Clinical Challenges; David Goldsmith,Adrian Covic,Jonas Spaak Book 2015 Springer International Publishing Switzerland 2015 An
描述.????????In modern medicine, the aging population is prone to many simultaneous cardiovascular (CV) risk?factors?which often produce co-incident pathology. Nowhere is this more obvious than in the interaction between Chronic Kidney Disease (CKD), Diabetes and diverse CV diseases (CVD). This is a complex and challenging area, as the presence of CKD/diabetes promotes CVD while also complicating its treatment. The emergence of CKD as a public health priority is one of the most challenging problems of modern medicine. It is now solidly established that renal dysfunction portends a high risk for cardiovascular disease. Cardiovascular diseases remains the main cause of death in western societies and the amplification of the death risk conveyed by coexisting CKD, even though still poorly understood, appears considerable. The bidirectional link that associates renal and cardiovascular diseases, the high risk of the death signalled by their coexistence and the considerable epidemiological burden imposed by this link is at the basis of the emergence of a new discipline aiming at making the borders between nephrology and cardiovascular medicine even more permeable than before. The term Cardio
出版日期Book 2015
關(guān)鍵詞Anticoagulation; Arrhythmia; Arterial Stiffening; CKD; CVD; Calcification; Cardio-renal; Cardiovascular Dis
版次1
doihttps://doi.org/10.1007/978-3-319-09162-4
isbn_softcover978-3-319-34870-4
isbn_ebook978-3-319-09162-4
copyrightSpringer International Publishing Switzerland 2015
The information of publication is updating

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Anticoagulation in CKDrdial infarction, prosthetic valve replacement, and prevention and treatment of venous thromboembolism. The risk of stroke in patients with stage 3 CKD and end-stage renal disease (ESRD) is higher than that of the general population and, in combination with atrial fibrillation, necessitates oral ant
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MRA Inhibition in CKD: More Than Salt and Watermost important causes of CKD in developed countries are diabetic nephropathy and renovascular disease/hypertension. Despite a proven favorable effect of RAAS blockade on short-term parameters (blood pressure, proteinuria) as well as long term outcome (slower decline of GFR), progression of chronic k
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Uric Acid, Allopurinol: The Cardio-Renal Silver Bullet?scular events. Uric acid is the end product of purine metabolism that circulates in the plasma at concentrations varying from 2 to 10 mg/dl or higher. Purine nucleotides are derived from both exogenous (alimentary intake, especially animal proteins) and endogenous sources (. molecule synthesis and n
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FGF23 and Phosphate: Two Cardiovascular Toxins with Distinct Toxicity Profiles?n integral element of CRS-4 is dysregulated mineral metabolism including altered serum levels of calcium and inorganic phosphorus (Pi). Downstream biochemical consequences entail remarkable elevations in the principal hormones regulating serum calcium and Pi, including parathyroid hormone (PTH), and
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Effect of Vitamin D on Endothelial Function and Blood Pressurers and is increasingly associated with non-traditional risk factors [2]. Vitamin D deficiency is a non-traditional risk factor for cardiovascular events which becomes more relevant with declining renal function. At the same time, the deficiency of active vitamin D plays a major role in the mineral-b
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