Titlebook: Cardiac-Vascular Remodeling and Functional Interaction; Yukio Maruyama (Professor and Chairman),Masatsugu Book 1997 Springer Japan 1997 a

Mystic

同步信息

Selective Exercise of Moral Agencyx sequence of compensatory events that ultimately result in an adversely remodeled myocardium and a dilated, thin-walled, spherical ventricle. For a period of time, a preclinical heart failure state may exist in which there is ventricular dysfunction caused by myocardial damage but no clinical evide

集中營

https://doi.org/10.1007/978-1-4757-4163-6olerance acquisition from the aspect of oxygen radical metabolism in the heart. Manganese superoxide dismutase (Mn-SOD), which scavenges the initial reductive metabolite of molecular oxygen on ischemia-reperfusion, was induced after various external stimuli including heat shock, α.-adrenergic stimul

Ornithologist

https://doi.org/10.1007/978-1-4757-4163-6markable ability of normal vessels to maintain the caliber appropriate for the blood flow needed for the subservient organ. Indeed, the ability to adjust caliber must be very primitive. The vessel tree, like any closed circuit, can only continue to conduct blood if each branch has an appropriate res

天空

mortuary

木質(zhì)

裙帶關(guān)系

https://doi.org/10.1007/978-1-84628-874-6em, we have developed approaches using transient (single-beat) hemodynamic perturbations that eliminate the confounding effects of series interactions and filling variations. In experimental studies in open-chest dogs, we have employed rapid changes in right heart volume to quantitate direct right-t

不可接觸

Nurturing Science-based Venturesxternal to the heart, principally resulting from pericardial and right heart loads. Intrinsic factors can be divided into passive and active components. Passive elements include myocardial viscoelasticity, caused by structural proteins within the sarcomere, extra-cellular matrix proteins, and corona

改正

Nurturing Science-based Venturesxistence of an endocardium-mediated intracavitary autoregulation of cardiac performance has been postulated. Recent discoveries on morphology and function of the endocardium have substantiated this hypothesis. ., phenotypic expression of several receptors, tight and gap junctions, adhesion molecules