Titlebook: Cardiac Preexcitation Syndromes; Origins, Evaluation, David G. Benditt (Associate Professor of Medicine) Book 1986 Martinus Nijhoff Publish

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Amilal Bhat,Mahakshit Bhat,Nikhil Khandelwal in severity from mild to life-threatening. Therefore, many patients with preexcitation syndromes and tachyarrhythmias will require treatment. The most appropriate form of therapy for these patients will depend upon the particular mechanism of the tachyarrhythmia. Although invasive electrophysiologi

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Irene Paraboschi,Massimo Garriboliave been extensively studied [1–10]. The most common arrhythmia in this patient population is the reciprocating tachycardia utilizing the normal pathway in one direction and the accessory pathway in the other. Less commonly, the accessory pathway may constitute both the anterograde and retrograde li

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Pharyngoesophageal Interrelationships,re insulated to cardiac impulses. An atrial impulse is conveyed to the ventricles by way of the normal AV conduction pathway — the AV node-His-Purkinje system [1,2]. The AV node possesses two unique electrophysiologic properties and functions as a protective barrier [3]: (1) delaying intranodal cond

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Emmett T. Cunningham Jr.,Bronwyn Jonest was limited to fibers connecting the His bundle to the septum but this was soon broadened to include fibers connecting the atrioventricular (AV) node to the septum. Although the existence of these has been confirmed anatomically [2–25], their functional significance has remained controversial [26–

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Radiation in Videorecorded Fluoroscopy, nature of this tachycardia the arrhythmia was named “permanent junctional reciprocating tachycardia” (PJRT). Although the clinical characteristics and electrocardiographic findings of PJRT have been confirmed following the original reports of the French authors, controversy has persisted for many y

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Epilepsy and the Cortex Anatomy,te all or part of the ventricular myocardium earlier than would have been expected had AV conduction been restricted to the normal cardiac conduction system [1–4]. Despite earlier doubts [5–7], it is now generally agreed that most forms of ventricular preexcitation are best accounted for by the pres