Titlebook: Cardiac Pacing; Proceedings of the V K. Steinbach Conference proceedings 1983 Dr. Dietrich Steinkopff Verlag, GmbH & Co. KG, Darmstadt 1983

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978-3-642-72369-8Dr. Dietrich Steinkopff Verlag, GmbH & Co. KG, Darmstadt 1983

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Suhas Govind Joshi,Tone Bratteteigd evenhanded selection of the papers to be presented at the congress. Each abstract was reviewed by three internationally recognized specialists in their field using a scoring system. The individual reviewers had been trained prior to the review process by completing a test review. Among the 573 pap

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Torben Tambo,Nikolaj Dybdal Clausen contraction. Previous work has shown that abnormal excitation of the left ventricle, such as occurs during ventricular pacing affects baseline hemodynamics, i.e. cardiac output and arterial pressure are depressed, while filling pressure rises (1,2). This depressant effect on myocardial performance

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Torben Tambo,Nikolaj Dybdal Clausen coronary artery. Isochronal maps of ventricular activation were constructed from close bipolar electrograms recorded from the entire epicardial surface and selected intramural sites utilizing a computerized multiplexing technique. The induced tachycardias were due to reentrant activation in the sur

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Nordic Contributions in IS Researchr tachyarrhythmias (VTs) using programmed pacing. Dogs were studied open chest under pentobarbital anesthesia both before and after either 0.2 (N = 5), 0.4 (N = 5) or 0.8 mg/kg (N = 5) intravenous diltiazem infused over 5 mimutes. Two doses were studied in 4 dogs. The maximal changes in heart rate,

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Bendik Bygstad,Hans-Petter Aanby,Jon Idented between 17 and 78 years, measuring in each of them effective (ERP) and functional (FRP) refractory periods at 3 sites of the right atrium (high, middle and low in the lateral wall) at the same driven frequency (120/min). Two threshold stimuli of 2 msec duration were applied. Dispersion of refrac

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Nordic Contributions in IS ResearchTypical SNE (TSNE), characterized by low-frequency, anatomically localized pre-P wave potentials (Reiffel et al.) were obtained in 12 of 18 patients (pts) with normal sinus node function (group I) and 15 of 26 pts with sick sinus syndrome (SSS) (group II). In 9 of 15 SSS pts but only 2 or 12 in grou