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Titlebook: Cardiac Ischemia: From Injury to Protection; Bohuslav O?t’ádal,Franti?ek Kolá? Book 1999 Springer Science+Business Media New York 1999 ada

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發(fā)表于 2025-3-25 06:43:16 | 只看該作者
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發(fā)表于 2025-3-25 10:03:33 | 只看該作者
Myocardial Vasculature during Ischemia and Reperfusion,ulation. There is now considerable evidence that ischemia can precipitate adverse structural and functional alterations in blood vessels. The extent to which, and the rate at which, vascular tissue can recover from the adverse consequences of ischemia is still unclear. Furthermore, there are differi
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發(fā)表于 2025-3-25 13:33:44 | 只看該作者
Cardiac Protection,ygen deprivation (Fig. 25). It is, therefore, not surprising that the interest of many experimental and clinical cardiologists during the past 35 years has been focused on the question of how cardiac tolerance to oxygen deprivation might be increased. The relatively short history of this endeavour r
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1566-0753 econditioning. The last part of the book dealswith comparative and ontogenetic aspects of cardiac sensitivity tooxygen deprivation; this chapter also summarizes the ontogeneticdifferences and limitations in endogenous and exogenous protection ofthe ischemic/hypoxic heart.978-1-4419-5105-2978-1-4757-3025-8Series ISSN 1566-0753
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發(fā)表于 2025-3-26 19:57:27 | 只看該作者
https://doi.org/10.1007/978-0-387-73717-1fy the detrimental consequences of ischemia and reperfusion. Thus, the microcirculation should represent an important target for innovative pharmacological interventions, particularly in the context of clinical conditions such as thrombolysis and cardiac surgery.
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