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Titlebook: Cancer Therapeutic Targets; John L. Marshall Reference work 2017 Springer Science+Business Media New York 2017 Anti-angiogenic Targets.HER

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發(fā)表于 2025-3-21 16:28:32 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Cancer Therapeutic Targets
編輯John L. Marshall
視頻videohttp://file.papertrans.cn/222/221210/221210.mp4
概述Offers a valuable reference work on cancer therapies for all levels of expertise.Structures information on cancer therapeutic targets, with regular updates.Provides expert editors’ overviews for each
圖書(shū)封面Titlebook: Cancer Therapeutic Targets;  John L. Marshall Reference work 2017 Springer Science+Business Media New York 2017 Anti-angiogenic Targets.HER
描述.In the past decade, we have experienced an explosion of new information about cancer therapeutic targets. Many of the targets have been validated by the discovery and approval of new medicines which have been approved for the treatment of cancer. On the heels of these successes, innumerable new targets and new potential therapeutics are being developed by many different groups including government agencies, pharmaceutical companies, biotechnology companies, academic institutions, and individual investigators. Understanding the expanding "universe" of cancer therapies is therefore becoming impossible and no single source exists which serves as a reference for the involved parties. Further, the interested parties have vastly different areas of expertise, from focused laboratory based science, to clinical research, to corporate and regulatory oversight. .The text would be updated every two years, more often depending on pace of change, interest and sales. While useful online, this reference book would likely be kept in hard copy as well..
出版日期Reference work 2017
關(guān)鍵詞Anti-angiogenic Targets; HER2/EGFR Targets; Nuclear Receptors; Hormone Receptors; Heme
版次1
doihttps://doi.org/10.1007/978-1-4419-0717-2
isbn_ebook978-1-4419-0717-2
copyrightSpringer Science+Business Media New York 2017
The information of publication is updating

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Schlussfolgerungen und Ausblick,tor (HER) family, which includes EGFR (or HER1), HER2, HER3, and HER4. EGFR is expressed in a variety of cell types, but primarily cells of epithelial, mesenchymal, and neuronal origin. It has been shown to be critical in the development of epithelial cancers, and EGFR overexpression or constitutive action can promote angiogenesis and metastasis.
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CTLA-4This is a brief summary of the biologic basis for use of CTLA-4 blockade in the treatment of cancer, specifically melanoma and renal cell carcinoma. Included is the biology of CTLA-4, the preclinical and clinical studies to date. Lastly, the future goals and directions of CTLA-4 are discussed including combination with newer immunotherapies.
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EGFR, Immunologytor (HER) family, which includes EGFR (or HER1), HER2, HER3, and HER4. EGFR is expressed in a variety of cell types, but primarily cells of epithelial, mesenchymal, and neuronal origin. It has been shown to be critical in the development of epithelial cancers, and EGFR overexpression or constitutive action can promote angiogenesis and metastasis.
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發(fā)表于 2025-3-22 20:19:43 | 只看該作者
https://doi.org/10.1007/978-1-4419-0717-2Anti-angiogenic Targets; HER2/EGFR Targets; Nuclear Receptors; Hormone Receptors; Heme
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