Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus

波動(dòng)

https://doi.org/10.1007/978-3-531-91917-1n, 1968; Colomo & Rahamimoff, 1968; Lev Tov & Rahamimoff, 1980). Both the influx of calcium ions into the depolarized neuronal axon and their extrusion from the axoplasm, are affected by extracellular sodium.

glacial

On the Voltage Dependence of Neurotransmitter Releasef release results from an increase in intracellular Ca. concentrations near critical sites. Correspondingly, termination of release must result from a drop in Ca. concentration near these critical sites to levels insufficient to support release (5,6,7).

Yag-Capsulotomy

Antimicrobial

BOAST

含水層

Wilfried von Bredow,Thomas J?gere potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and intracellular calcium concentrations (see 20; 28; 40).

嘲弄

Resting Calcium Levels and Evoked Release at the Neuromuscular Junction This single addition substantially alters the behavior of the model and makes it necessary to reinterpret many previous measurements. We have presented the mathematics for the model elsewhere (2, 3). Here we will give a more general description of our model, its application to experiments, and some directions for future research.

征服

Calcium Independent Quantal Transmitter Release at the Neuromuscular Junctione potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and intracellular calcium concentrations (see 20; 28; 40).

FAZE

Influx