Titlebook: Brain Plasticity; Development and Agin Guido Filogamo,Antonia Vernadakis,Paola S. Timiras Book 1997 The Editor(s) (if applicable) and The A

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Ischemia

IKLTSA: An Incremental Kernel LTSA Methodview shall deal with these aspects with the main emphasis on glutamate uptake and metabolism. It has been generally accepted for a number of years that astrocytes play a very important role in these processes (see, Schousboe, 1981) and therefore emphasis will be placed on a discussion of the role of

countenance

Gayane Shalunts,Gerhard Backfriedng the last 100 years, there has been an unequivocal lengthening of the life expectancy of individuals, accompanied by a dramatic increase in the rate of neurodegenerative Parkinson’s and Alzheimer’s diseases (Lilienfeld et al., 1990; Rajput, 1992). Meanwhile, the threshold age for the onset of Park

investigate

Genes Controlling Neural Fate and Differentiationf many of the extracellular signalling factors and intracellular proteins that direct cell fate. In this chapter we a) present a brief overview of recent progress made in defining positive and negative regulators of neurogenesis in vertebrates and their similarities with invertebrate organisms, b) d

evaculate

Neuronal Plasticity in Development: Lessons from Ethanol Neurotoxicity during Embryogenesisor static. They found that the migratory destination of autonomic neuroblasts of neural crest origin determines their ultimate neurotransmitter phenotype. Furthermore, they demonstrated that exposure of neural crest-derived presumptive adrenergic neuroblasts to cells from another germ cell layer, sp

FLINT

Plasticity in Astrocytic Phenotypesstrocyte, astrocytes undergo dynamic shape-function changes. Most intermediate and differentiated phenotypes of astrocytes are characterized by expression of specific cytoskeletal proteins and the acquisition of specific shape (reviewed in Cameron and Rakic, 1991). After final positioning and cell p

MAIM

Neuron-Glia Cross Talk in Rat Striatum after Transient Forebrain Ischemia)-containing interneurons are resistant to the ischemic insult (Pulsinelli et al. 1982, Grimaldi et al. 1990). In a few instances, such as in the case of SS and NPY immunoreactivity (IR), the initial loss is followed by full recovery within 7 (SS) or 40 (NPY) days post-ischemia (Grimaldi et al. 1990

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外貌

Role of Astrocytes in Glutamate Homeostasisview shall deal with these aspects with the main emphasis on glutamate uptake and metabolism. It has been generally accepted for a number of years that astrocytes play a very important role in these processes (see, Schousboe, 1981) and therefore emphasis will be placed on a discussion of the role of