Titlebook: Bone Regulatory Factors; Morphology, Biochemi Antonio Pecile (Professor of Pharmacology),Benedet Conference proceedings 1990 Springer Scien

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Stevan A. Gonzalez,Robert P. Perrillore of bone in patients with hyperparathyroidism (Hunter and Turnbull, 1932). The main pathological changes which lead to this include an increase in number (and apparently activity) of osteoclasts, marrow fibrosis, and sometimes the formation of expanded cyst-like lesions, or brown tumours. Although

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https://doi.org/10.1007/978-3-030-70773-6 Queener & Bell, 1975; Maclntyre et al, 1988; Wimalawansa & Maclntyre, 1989b). Initial studies on sheep suggested the parathyroid gland as the source of CT (Copp & Henze, 1964). Later, the source of this calcium lowering factor was identified as the thyroid gland (Foster et al, 1964a). CT is synthes

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Biliary Hamartomas (von Meyenberg Complexes)f behaviour. The peptides directed by the calcitonin gene have been particularly thoroughly studied but immunoreactive PTH material has also been discovered in the brain and has also been suggested to be a neuromodulator within the nervous system. We will consider first the peptides directed by the

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The Vitamin D Endocrine System and Bone,osphorus metabolism. Along with the two peptide hormones PTH and calcitonin, vitamin D is responsible for the minute-to-minute as well as the day-to-day establishment and maintenance of calcium homeostasis.

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