Titlebook: Biology of Menopause; Francis L. Bellino Conference proceedings 2000 Springer Science+Business Media New York 2000 Gonadotropin.bone.cells

音樂會

Frührehabilitation an der Intensivstation). Follicular reserve is depleted at a steady rate until the age of 37. After that age, the rate of follicle depletion accelerates until the follicle supply is exhausted (2). In parallel to this acceleration, baseline FSH levels rise and the fecundity rates decline (3–6). Studies on ovarian aging, t

Trypsin

Darstellung der Schaltungsunterlagen, (NT-4/5)] have been implicated in the development, survival, plasticity, and aging of neurons in mammalian forebrain regions that subserve cognitive functions. Neurons in overlapping forebrain regions of both sexes coexpress estrogen and neurotrophin receptors, and they are the sites of estrogen an

commodity

,Affektive St?rungen (F30–F39),crease in mortality (1). Of particular interest to neurodegenerative disease, ERT correlates with a decreased incidence of Alzheimer’s disease (AD) (2,3), reducing the onset of the disease by as much as 10 years in one study (3). Further, several small clinical studies support a role for estrogen th

nuclear-tests

mastopexy

maladorit

Kompendium Praktische Psychiatried growth, bones are sculpted in order to achieve their shape and size by the removal of bone from one site and deposition at a different one; this process is called . After maturity is reached, regeneration continues in the form of a periodic replacement of old bone with new at the same location. Th

含鐵

,Affektive St?rungen (F30–F39),erence in coronary heart disease (CHD) mortality rates is largest around the time of the average age of menopause in Caucasians and to a lesser extent in African-Americans (1). Comparisons of pre- and postmenopausal women categorized into 5-year age groups show higher rates of cardiovascular disease

progestin

比賽用背帶

,Entwicklungsst?rungen (F80–F89),ause are associated with increased risk of CHD (1), although estrogen replacement therapy (ERT) reduces the risk of CHD by about 50% in postmenopausal women (2–4) and the amount of atherosclerosis by about 50% in animal models (5–8). Even though there is overwhelming evidence that estrogen monothera

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,Intelligenzminderung (F70–F79),luence on morbidity and mortality. Epidemiological data indicate that women in their reproductive years have a much lower incidence of coronary disease than men of similar age, an advantage that diminishes rapidly with the onset of menopause (1,2). These studies demonstrate a direct correlation betw