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Titlebook: Biochemistry of Apoptosis and Autophagy; Lorrie A. Kirshenbaum Book 2022 Springer Nature Switzerland AG 2022 Apoptosis.Autophagy.Cell Deat

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發(fā)表于 2025-3-21 17:15:38 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Biochemistry of Apoptosis and Autophagy
影響因子2023Lorrie A. Kirshenbaum
視頻videohttp://file.papertrans.cn/187/186692/186692.mp4
發(fā)行地址Covers all aspects of apoptosis.Chapters submitted from world renowned researchers.Unique in defining the relationship between the regulatory mechanisms
學(xué)科分類Advances in Biochemistry in Health and Disease
圖書(shū)封面Titlebook: Biochemistry of Apoptosis and Autophagy;  Lorrie A. Kirshenbaum Book 2022 Springer Nature Switzerland AG 2022 Apoptosis.Autophagy.Cell Deat
影響因子One of the most intriguing and compelling issues to impact contemporary biology to date is the concept that cell death is genetically regulated. Observations by Kerr and Wyllie, made more than 30 years ago on the basis of distinct morphological criteria, markedly distinguished apoptosis from classical cell death by necrosis. Apoptosis is a highly regulated, evolutionary conserved, genetic program of cell death essential for normal development and tissue homeostasis. The discovery of apoptosis as a regulated event and potentially amenable to therapeutic interventions has generated considerable excitement because it meant that disease entities resulting from either too much, or too little, apoptosis could be potentially cured with new therapies that target apoptosis..While there is little doubt that necrosis induced by massive cellular trauma is likely an unregulated event, several lines of investigation have challenged the dogma that necrotic cell death ismerely unregulated. Emerging data has shifted the paradigm in our thinking about necrosis as a regulated event..Autophagy is another cellular process that has received considerable attention over the past two decades and its remark
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978-3-030-78801-8Springer Nature Switzerland AG 2022
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Lorrie A. KirshenbaumCovers all aspects of apoptosis.Chapters submitted from world renowned researchers.Unique in defining the relationship between the regulatory mechanisms
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Advances in Biochemistry in Health and Diseasehttp://image.papertrans.cn/b/image/186692.jpg
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https://doi.org/10.1007/978-3-322-90036-4rodents. Individual cardiomyocytes are thus quite long-lived and must be able to continuously adapt to physiologic and pathophysiologic stresses. Stress adaptation often entails the renewal of intracellular constituents, as exemplified by the changes in enzymatic activity which accompanies shifts in
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