Titlebook: Biochemical Regulation of Myocardium; Roland Vetter,Ernst-Georg Krause Book 1996 Kluwer Academic Publishers 1996 ATP.Calcium.Glycogen.Lipi

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β2-Adrenoceptor activation by zinterol causes protein phosphorylation, contractile effects and relaxused positive inotropic and lusitropic (i.e. reduction of t. of relaxation) effects with EC. values of 3 and 2 nM, respectively. The zinterol-evoked effects were unaffected by the β.AR-selective antagonist CGP 20712A (300 nM) but blocked surmountably by the β.AR-selective antagonist ICI 118551 (50 n

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Early after-depolarisations induced by noradrenaline may be initiated by calcium released from sarcouration and DATs resulted in subthreshold delayed after-depolarisations (DADs). When sarcolemmal K. conductance was decreased by tetraethylammonium (TEA) in the patch electrode or 20 μM BaCl. in the Tyrode solution, EATs initiated early after depolarizations (EADs) and DATs initiated suprathreshold

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Cardiac pump function of the isolated rat heart at two modes of energy deprivation and effect of adrt, a curve relating LV filling volume and end-diastolic pressure was shifted up and to the left much more prominently after . treatment. Heart rate was decreased by 24% only in amytal-treated group. Results indicate that a decreased myocardial distensibility is a dominating feature in the acute card

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Heart glycogen content and isoprenaline-induced myocardial lesionsenylyl cyclase. On the other hand, these parameters did not differ between IR and IS rats in spite of the presence of significant differences in the resistance to the development of IML and in heart glycogen content in these two rats strains..These results suggest that genetically determined differe