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Titlebook: Bacterial Invasion into Eukaryotic Cells; Subcellular Biochemi Tobias A. Oelschlaeger,J?rg Hacker Book 2000 Springer Science+Business Media

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發(fā)表于 2025-3-25 05:20:44 | 只看該作者
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發(fā)表于 2025-3-25 08:37:57 | 只看該作者
Interaction of Yersinia with Host Cellser and Aber, 1989). Although these three pathogens have different entry routes and cause different diseases, they share a remarkable tropism for the lymphoid tissues of their host, where they are able to resist the non-specific immune defense mechanisms.
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發(fā)表于 2025-3-25 11:56:16 | 只看該作者
Edward Peter Stringham,Louis B. Salz of the rhizobial genes (.) required for nodulation (Fellay .., 1995). Many . loci are involved in the synthesis and secretion of Nod-factors, a family of complex lipo-chito-oligosaccharides (Hanin .., 1998b; Dénarié .., 1996). Nod-factors initiate division of nodule meristems and permit entry of rhizobia into the host-plant (Relic .., 1994).
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發(fā)表于 2025-3-25 17:10:46 | 只看該作者
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發(fā)表于 2025-3-26 15:28:39 | 只看該作者
Invasion of Mammalian and Protozoan Cells by ,lmark of Legionnaires’ disease is the intracellular replication of . in the alveolar spaces. At least another 39 species of . have been identified, some of which are associated with disease while others are environmental isolates and whether they can cause disease is not known. . is responsible for
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發(fā)表于 2025-3-26 19:23:58 | 只看該作者
https://doi.org/10.1007/978-3-319-05915-0with an eukaryotic receptor to induce a signal transduction cascade leading ultimately to the cytoskeletal rearrangements necessary for internalization. Of special interest are the receptors and intermediary host molecules which transduce the initial bacterial signal through the host cell thereby ac
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