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Titlebook: Bacterial Infection: Close Encounters at the Host Pathogen Interface; Peter K. Vogt,Michael J. Mahan Book 1998 The Editor(s) (if applicabl

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31#
發(fā)表于 2025-3-27 00:28:23 | 只看該作者
Handbook on Hospital Planning & Designing siderophore-mediated mechanism (. 1980, 1981, 1991, 1995; . 1987) and those that utilize a mechanism involving transferrin-bound iron (. and . 1989; . and . 1992). The culmination of these studies is a broad literature describing the molecular and biochemical basis for high-affinity iron transport.
32#
發(fā)表于 2025-3-27 05:04:04 | 只看該作者
33#
發(fā)表于 2025-3-27 09:16:15 | 只看該作者
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發(fā)表于 2025-3-27 12:39:03 | 只看該作者
Victoria Herrmann,Lillian Hussongollowing is a review of the biochemistry, structure, and mechanisms of pathogenicity of the PTSAg and the shared and unique properties of each. The properties of other relevant superantigenic proteins such as the staphylococcal exfoliative toxins (ETA, ETB) will also be discussed.
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發(fā)表于 2025-3-27 15:25:50 | 只看該作者
36#
發(fā)表于 2025-3-27 20:07:31 | 只看該作者
Ignacio Ferrero,Andrea Roncella,Marta Rocchin host tissues. In this review, we will describe two complementary genetic strategies developed in . that allow the isolation of bacterial genes induced or required during infection. The identification of such genes and the products they encode provides a means to understand their contributions to infection, virulence, and pathogenesis.
37#
發(fā)表于 2025-3-27 22:00:25 | 只看該作者
Michael Delaunay,Mathieu Landriaultan active role for the innate immune system in the demise of the anthrax victim. Many of the molecular factors and events in the cascade of lethal events during anthrax infections have now been identified. Other recent overviews of anthrax pathogenesis and toxins include those by . (1986), . (1990), . (1995), and . and . (1997).
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發(fā)表于 2025-3-28 03:00:40 | 只看該作者
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發(fā)表于 2025-3-28 10:04:41 | 只看該作者
In Vivo Gene Expression: Contributions to Infection, Virulence, and Pathogenesis,n host tissues. In this review, we will describe two complementary genetic strategies developed in . that allow the isolation of bacterial genes induced or required during infection. The identification of such genes and the products they encode provides a means to understand their contributions to infection, virulence, and pathogenesis.
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