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Titlebook: Bacterial Adhesion to Cells and Tissues; Itzhak Ofek,Ronald J. Doyle Book 1994 Chapman & Hall, Inc. 1994 bacteria.bacterial adhesion.bacte

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21#
發(fā)表于 2025-3-25 06:14:53 | 只看該作者
22#
發(fā)表于 2025-3-25 08:10:47 | 只看該作者
23#
發(fā)表于 2025-3-25 11:51:53 | 只看該作者
Book 1994d virulence factors is involved in thisprocess, which recent research has greatly illuminated. Thiscomprehensive and authoritative volume discusses the specific celland tissue-specific affinities of pathogenic microorganisms,including bioinorganic surfaces such as teeth, and is an essentialreference
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發(fā)表于 2025-3-25 19:38:05 | 只看該作者
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發(fā)表于 2025-3-25 21:25:03 | 只看該作者
26#
發(fā)表于 2025-3-26 04:06:25 | 只看該作者
Jun Liu,Simone Ciampi,Andrew Antonylectins is provided in Chapter 9. In this chapter information is presented concerning the occurrence and specificities of some bacterial surface lectins and their role in infection. Many bacterial lectins have not yet been defined as adhesins. A comprehensive review of bacterial lectins is found in the book edited by Mirelman (1986).
27#
發(fā)表于 2025-3-26 06:02:17 | 只看該作者
Jun Liu,Simone Ciampi,Andrew Antonye, termed lectinophagocytosis, is based on recognition between surface lectins on one cell and carbohydrates on the opposing cell. The second mechanism involves protein-protein interactions via the Arg-Gly-Asp (RGD) sequence. The final mechanism involves hydrophobic interactions between the two cell types.
28#
發(fā)表于 2025-3-26 11:08:51 | 只看該作者
Relationship Between Bacterial Cell Surfaces and Adhesins,with special emphasis on macromolecules involved in adhesion, is given. More comprehensive discussions of the bacterial cell surface are provided elsewhere (Rogers et al., 1980; Nikaido and Vaara, 1985; Krell and Beveridge, 1987; Doyle and Sonnenfeld, 1989; Handley, 1990; Hancock, 1991; Irvin, 1990; Gilbert et al., 1991).
29#
發(fā)表于 2025-3-26 15:05:36 | 只看該作者
30#
發(fā)表于 2025-3-26 20:01:52 | 只看該作者
Interaction of Bacteria with Phagocytic Cells,e, termed lectinophagocytosis, is based on recognition between surface lectins on one cell and carbohydrates on the opposing cell. The second mechanism involves protein-protein interactions via the Arg-Gly-Asp (RGD) sequence. The final mechanism involves hydrophobic interactions between the two cell types.
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